Mitochondrial remodelling

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Lab affiliation

Associate Professor Dept. of Pathology and Laboratory Medicine HSRF 218, University of Vermont Larner College of Medicine Burlington, Vermont, 05405, USA (1)
Biological Chemistry and Drug Discovery, School of Life Sciences, University od Dundee, Dundee (1)
Biomedical Research Centre, University Hospital Hradec Kralove, Hradec Kralove, Czech Republic (1)
Biosciences Institute, Faculty of Medical Sciences, Newcastle University, Newcastle upon Tyne, United Kingdom (1)
CSIR-Centre for Cellular and Molecular Biology, Uppal Road, Hyderabad - 500007, India (1)
Center for Physiology and Pathophysiology, Institute of Vegetative Physiology, University of Köln, Köln, Germany Cologne Excellence Cluster on Cellular Stress Responses in Aging-associated Diseases (CECAD), University of Cologne, Cologne, Germany (lab head) (1)
Centre for Medical Research, The University of Western Australia, QEII Medical Centre, Nedlands, Western Australia 6009, Australia (1)
Charles University, Faculty of Science, Department of Parasitology, BIOCEV, Vestec 252 50, Czech Republic. (1)
Dame Kate Campbell Fellow Department of Surgery| Melbourne Medical School Department of Anatomy and Physiology| School of Biomedical Sciences Faculty of Medicine, Dentistry and Health Sciences Medical Building (#181), Grattan Street The University of Melbourne, Victoria 3010 Australia P: +61 3 9035 5464 E: apebay@unimelb.edu.au W: https://medicine.unimelb.edu.au/school-structure/surgery W: https://biomedicalsciences.unimelb.edu.au/departments/anatomy-and-physiology (1)
Department of Basic and Clinical Neuroscience, Institute of Psychiatry, Psychology and Neuroscience, Maurice Wohl Clinical Neuroscience Institute, King’s College London, 125 Coldharbour Lane, London SE5 9NU, U.K (1)
Department of Biochemistry and Pharmacology, Bio21 Molecular Science and Biotechnology Institute, The University of Melbourne, Melbourne, VIC, 3010, Australia 2Murdoch Children’s Research Institute, Melbourne, VIC 3052, Australia 3Victorian Clinical Genetics Services, Murdoch Children’s Research Institute, Melbourne, VIC, 3052, Australia (1)
Department of Biomedical Sciences, Jepps lab, University of Copenhagen, Denmark (1)
Department of Developmental Biology and Cancer Research,Hebrew University of Jerusalem, Israel (1)
Department of Physiology and Medical Physics, Royal College of Surgeons in Ireland, Dublin, Ireland. (1)
Director, Duke Molecular Physiology Institute, Professor Departments of Medicine (Division of Endocrinology, Metabolism, and Nutrition) and Pharmacology & Cancer Biology Duke University Medical Center (1)
Faculty of Medicine & Health, The University of Sydney, Camperdown, NSW 2006, Australia. (1)
Full Professor, Director Of Academic Centre - Conway Institute School of Public Health, Physiotherapy and Sports Science (1)
Genetics Section Molecular and Clinical Sciences St. George's, University of London Jenner 2.142B, Cranmer Terrace London SW17 0RE (1)
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IRCCS Fondazione Bietti, Rome, Italy (1)
Institut des Sciences du Sport de l'Université de Lausanne Bâtiment Synathlon 1015 Lausanne Switzerland (1)
Institute for Health and Sport (iHeS), Victoria University, Australia (1)
Institute of Healthy Ageing, UCL Department of Genetics, Evolution and Environment, London, WC1E 6BT (1)
Institute of Life Course & Medical Sciences Faculty of Health and Life Sciences University of Liverpool Liverpool UK (1)
Institute of Molecular and Cell Biology, Agency for Science, Technology and Research, TBP Lab (1)
King’s British Heart Foundation Centre of Research Excellence, King's College London, United Kingdom (1)
Laboratoire de Spectrométrie de Masse BioOrganique, Analytical Science department, IPHC, CNRS, Strasbourg University, UMR7178, Strasbourg, France (1)
Liverpool John Moores University (1)
MRC-Laboratory of Medical Sciences (1)
Max Planck Institute for Biology of Ageing (1)

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Genotype to phenotype: Diet-by-mitochondrial DNA haplotype interactions drive metabolic flexibility and organismal fitness

Diet may be modified seasonally or by biogeographic, demographic or cultural shifts. It can differentially influence mitochondrial bioenergetics, retrograde signalling to the nuclear genome, and anterograde signalling to mitochondria. All these interactions have the potential to influence the freque...

2018-11-07 | MTBLS724 | MetaboLights

Exercise-generated β-aminoisobutyric acid (BAIBA) reduces cardiomyocytes metabolic stress and apoptosis caused by mitochondrial dysfunction through the miR-208b/AMPK pathway

OBJECTIVE: To explore the cardioprotective effects of exercise-derived β-aminoisobutyric (BAIBA) on cardiomyocyte apoptosis and energy metabolism in a rat model of heart failure (HF).

METHODS: In male Sprague-Dawley rats (8-week-old), myocardial infarction ...

2022-03-08 | MTBLS3927 | MetaboLights

Mitochondrial stress drives skeletal muscle remodelling following sprint-interval exercise.

Mitochondria play a central role in cellular metabolism and are crucial for maintaining cellular health. Exercise is a proven intervention to enhance mitochondrial function and prevent non-communicable diseases, yet the molecular mechanisms driving exercise-induced mitochondrial remodelling remain u...

ORGANISM(S): Homo sapiens (Human)

2026-01-12 | PXD059070 | Pride

Adaptive ER stress promotes mitochondrial remodelling and longevity through PERK-dependent MERCS assembly

PRJNA1204992 | ENA

The APC/C activator Cdh1p plays a role in mitochondrial metabolic remodelling in yeast

Cdh1p is one of the two substrate adaptor proteins of the anaphase promoting complex/cyclosome (APC/C), an ubiquitin ligase that regulates proteolysis during cell cycle. In this work, using a proteomic approach, we found 135 mitochondrial proteins whose abundance was significantly altered in the cdh...

ORGANISM(S): Saccharomyces cerevisiae (Baker's yeast)

2023-02-24 | PXD039879 | Pride

Proteomics Yeast Mitochondria Yap1p Cdh1p

Respiratory chain signalling is essential for adaptive remodelling following cardiac ischaemia.

Not available

S-EPMC7131948 | biostudies-literature

Mitochondrial implications in human pregnancies with intrauterine growth restriction and associated cardiac remodelling.

Not available

S-EPMC6533501 | biostudies-literature

MicroRNA-27b-3p down-regulates FGF1 and aggravates pathological cardiac remodelling.

Not available

S-EPMC9302889 | biostudies-literature