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TranscriptomicsGene expression profile after dexamethasone in chronic lymphocytic leukemia cells according to IGHV/ZAP-70 status
Glucocorticoids are part of the therapeutic armamentarium of chronic lymphocytic leukemia where it has been suggested that cells with unmutated IGHV genes exhibit higher sensitivity. The mechanisms by which glucorticoids are active in CLL are not well elucidated. We used microarrays to detail the gl...
ORGANISM(S): Homo sapiens 
2012-12-04 | E-GEOD-33135 | biostudies-arrayexpress
UnknownOral Administration of Vitamin D3 Prevents Corneal Damage in a Knock-Out Mouse Model of Sjogren's Syndrome.
Not available
S-EPMC9953695 | biostudies-literature
UnknownExtracellular acidity and ATP modulate ion currents in human cumulus cells indicating possible roles as metabolic sensors of the follicular microenvironment.
Not available
S-EPMC12849215 | biostudies-literature
UnknownThe PpPep2-Triggered PTI-like Response in Peach Trees Is Mediated by miRNAs.
Not available
S-EPMC11642718 | biostudies-literature
UnknownImpact of diffused versus vasculature targeted DNA damage on the heart of mice depleted of telomeric factor Ft1.
Not available
S-EPMC10726857 | biostudies-literature
UnknownDifferences in total and differential white blood cell counts and in inflammatory parameters between psychiatric inpatients with and without recent consumption of cannabinoids, opioids, or cocaine: A retrospective single-center study.
Not available
S-EPMC11615885 | biostudies-literature
UnknownDevelopment and activity of canine B7-H3-CAR.CIK lymphocytes against sarcomas: preclinical evidence and perspectives for human clinical translation.
Not available
S-EPMC12433426 | biostudies-literature
UnknownExamining cervical cancer screening adherence: how does healthism influence participation?
Not available
S-EPMC12513119 | biostudies-literature
TranscriptomicsHistone acetylation and DNA demethylation of T-cells result in an anaplastic large cell lymphoma-like phenotype.
A characteristic feature of anaplastic large cell lymphoma (ALCL) is the significant reduction of the T-cell expression program despite its T-cell origin, a finding very similar to the loss of B-cell identity of classical Hodgkin lymphoma (cHL). Previously we demonstrated that epigenetic mechanisms ...
ORGANISM(S): Homo sapiens 
2012-08-19 | E-GEOD-26101 | biostudies-arrayexpress
UnknownTargeting HDAC2-Mediated Immune Regulation to Overcome Therapeutic Resistance in Mutant Colorectal Cancer.
Not available
S-EPMC10093005 | biostudies-literature
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