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ProteomicsDetermination of pancreatic cyst fluids peptidome and proteome
Determination of pancreatic cyst fluids (PCF) peptidome and proteome in 5 distinct patient groups: malignant (CAs), intraductal papillary mucinous neoplasms (IPMNs), mucinous cystic neoplasms (MCNs), serous cystic neoplasms (SCNs), and pseudocysts (PCs). The PCF <5kDa fraction, referred as the degra...
ORGANISM(S): Homo sapiens (Human) 
2019-01-14 | PXD005248 | Pride
Human Pancreas Cyst fluids Degradome Proteome Lc-ms/ms 
ProteomicsHuman bladder cancer and colon cancer quatitative MS/MS
We analyzed a set of 40 independently collected tumor tissue/control samples using a label-free quantitation single pass LC-MS/MS approach. In this set, 10 samples originated from a colon tumor (i.e. AC) and 10 from control non-malignant tissue samples (NCA), as well as 10 from BC samples and 10 fro...
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
MSV000082886 | MassIVE
TranscriptomicsMolecular defense mechanisms of Barrett's metaplasia estimated by an integrative genomics
Barrett's esophagus is characterized by the replacement of squamous epithelium with specialized intestinal metaplastic mucosa. The exact mechanisms of initiation and development of Barrett's metaplasia remain unknown, but a hypothesis of successful adaptation against noxious reflux components has be...
ORGANISM(S): Homo sapiens 
2012-03-08 | E-GEOD-36223 | biostudies-arrayexpress
TranscriptomicsHepatic molecular alterations more than muscle's differentiate obese hyperphagic mice from those prolonged fed with the high-fat diet
Although mitochondrial dysfunctions are implicated in the pathogenesis of obesity, the molecular mechanisms underlying obesity-related metabolic abnormalities are still not well established. To acquire a comprehensive picture of mitochondrial molecular changes within metabolically active tissues, we...
ORGANISM(S): Mus musculus 
2013-05-01 | E-GEOD-43691 | biostudies-arrayexpress
TranscriptomicsModeling oncogenic signaling in colon tumors by multidirectional analyses of microarray data
Background. Most colorectal cancers (CRC) arise in a progression through adenoma to carcinoma phenotypes as a consequence of altered genetic information. Clinical progression of CRC may occur in parallel with distinctive signaling alterations. We designed multidirectional analyses integrating microa...
ORGANISM(S): Homo sapiens 
2010-08-01 | E-GEOD-20916 | biostudies-arrayexpress
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