Sort   by:  
 Page size 
TranscriptomicsAutotaxin and its product lysophosphatidic acid suppress brown adipose differentiation and promote diet-induced obesity in mice
Brown adipose tissue (BAT) is a thermogenic organ that dissipates stored energy as heat to maintain body temperature in infants and small mammals. This process may also provide protection from development of diet-induced obesity. We found that the bioactive lipid mediator lysophosphatidic acid (LP...
ORGANISM(S): Mus musculus 
2011-06-01 | E-GEOD-26442 | biostudies-arrayexpress
GenomicsMMSET alters EZH2 Binding in Myeloma Cells
We investigated EZH2 binding in the presence and absence of MMSET protein. MMSET overexpression in t(4;14)+ myeloma leads to global loss of H3K27 methylation and redistribution of EZH2 binding throughout the genome ChIP-seq for EZH2 in two cell types
ORGANISM(S): Homo sapiens 
2014-09-01 | E-GEOD-57632 | biostudies-arrayexpress
UnknownCa2+ entry through NaV channels generates submillisecond axonal Ca2+ signaling.
Not available
S-EPMC7380941 | biostudies-literature
UnknownFatal late-onset CAR T-cell-mediated encephalitis after axicabtagene-ciloleucel in a patient with large B-cell lymphoma.
Not available
S-EPMC8679677 | biostudies-literature
TranscriptomicsDeep Sleep and Parietal Cortex Gene Expression Changes are Related to Cognitive Deficits with Age
Background: Age-related cognitive deficits negatively affect quality of life and can presage serious neurodegenerative disorders. Despite sleep disruption’s well-recognized negative influence on cognition, and its prevalence with age, surprisingly few studies have tested sleep’s relationship to cogn...
ORGANISM(S): Rattus norvegicus 
2011-04-21 | E-GEOD-24515 | biostudies-arrayexpress
TranscriptomicsMicroarray analyses of laser-captured hippocampus reveal distinct gray and white matter signatures associated with incipient Alzheimer’s disease
Alzheimer's disease (AD) is a devastating neurodegenerative disorder that threatens to reach epidemic proportions as our population ages. Although much research has examined molecular pathways associated with AD, relatively few studies have focused on critical early stages. Our prior microarray stud...
ORGANISM(S): Homo sapiens 
2011-07-31 | E-GEOD-28146 | biostudies-arrayexpress
UnknownInsight into the Postbiotic Potential of the Autochthonous Bacteriocin-Producing Enterococcus faecium BGZLM1-5 in the Reduction in the Abundance of Listeria monocytogenes ATCC19111 in a Milk Model.
Not available
S-EPMC10745621 | biostudies-literature
UnknownSubcritical Water and Pressurised Ethanol Extractions for Maximum Recovery of Antioxidants from Orange Peel Herbal Dust with Evaluation of Its Pharmacological Potential Using In Silico and In Vitro Analysis.
Not available
S-EPMC12189749 | biostudies-literature
UnknownMatrix-assisted laser desorption/ionization time of flight mass spectrometry identification of Vibrio (Listonella) anguillarum isolated from sea bass and sea bream.
Not available
S-EPMC6860450 | biostudies-literature
ProteomicsMitochondrial folate one-carbon metabolism moderates Alzheimer’s disease pathology
Familial forms of AD cause a loss of neurons associated with the accumulation of toxic amyloid β peptide (Aβ) and mitochondrial dysfunction. We found that flies over expressing a toxic form of Aβ (Aβ-Arc) exhibit altered components of mitochondrial complex I and one-carbon (1C) metabolism.
ORGANISM(S): Drosophila melanogaster (Fruit fly) 
2025-05-06 | PXD040263 | Pride
Amyloid-beta Mitochondria Drosophila Alzheimer's disease 
Sort   by:  
 Page size