Tan A R AR - OmicsDI

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Homo sapiens (36)
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biostudies-arrayexpress (19)
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pride (5)
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CHEBI ID

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Metabolite Name

1-methylguanidine (1)
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1-palmitoyl-2-arachidonoyl-GPC (16:0/20:4) (1)
1-palmitoyl-2-oleoyl-GPC (16:0/18:1) (1)
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Transcription profiling by array (11)
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Lab affiliation

Department of Biology, Institute of Molecular Systems iiology, ETH Zürich, Switzerland (1)
Department of Radiation Oncology, College of Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA (1)
School of Bioscience and Bioengineering, South China University of Technology, Guangzhou, P. R. China (1)
University of Minnesota, Twin-Cities, Pharmacology Department (1)
University of Minnesota, Twin-cities Pharmacology Department (1)

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NKX3-1, a Novel Transcriptional Factor of AR, Promotes Prostate Cancer Cell Survival via RAB3B GTPase-mediated protein trafficking

Androgen receptor (AR) orchestrates an intricate transcriptional regulatory network that governs prostate cancer initiation, development and progression. To understand this network in detail, we generated genome-wide maps of AR occupancy by ChIP-seq in LNCaP cells. We found NKX3-1, an androgen-depen...

ORGANISM(S): Homo sapiens

2011-11-16 | E-GEOD-28264 | biostudies-arrayexpress

TMPRSS2-ERG, HDACs and EZH2 are involved in an AR-centric transcriptional circuitry that calibrates androgenic response for prostate cancer progression (ChIP-Seq data)

Deregulation of the Androgen Receptor (AR) transcriptional network is a common hallmark in prostate cancers. To achieve its precise transcriptional role, AR needs to co-operate specifically with a plethora of cofactors. In prostate cancers, AR transcription collaborators are frequently aberrantly ov...

ORGANISM(S): Homo sapiens

2012-04-26 | E-GEOD-28950 | biostudies-arrayexpress

TMPRSS2-ERG, HDACs and EZH2 are involved in an AR centric transcriptional circuitry that calibrates androgenic response for prostate cancer progression (gene expression after ERG KD)

ORGANISM(S): Homo sapiens

2012-04-26 | E-GEOD-35540 | biostudies-arrayexpress

TMPRSS2-ERG, HDACs and EZH2 are involved in an AR-centric transcriptional circuitry that calibrates androgenic response for prostate cancer progression (gene expression data)

ORGANISM(S): Homo sapiens

2012-04-26 | E-GEOD-28948 | biostudies-arrayexpress

Ligand-Dependent Enhancer Activation Regulated by Topoisomerase-I Activity

The discovery that enhancers are regulated transcription units, encoding eRNAs, has raised new questions about the mechanisms of their activation. Here, we report an unexpected molecular mechanism that underlies ligand-dependent enhancer activation, based on DNA nicking to relieve torsional stress f...

ORGANISM(S): Homo sapiens

2015-01-22 | E-GEOD-63202 | biostudies-arrayexpress

Dysregulation of Streptococcus pneumoniae zinc homeostasis breaks ampicillin resistance in a pneumonia infection model

Streptococcus pneumoniae is the primary cause of community-acquired bacterial pneumonia with rates of penicillin and multi-drug resistance exceeding 80% and 40%, respectively. The innate immune response generates a variety of antimicrobial agents to control infection including zinc stres...

2022-02-23 | MTBLS318 | MetaboLights

TMPRSS2-ERG, HDACs and EZH2 are involved in an AR-centric transcriptional circuitry that calibrates androgenic response for prostate cancer progression

This SuperSeries is composed of the following subset Series: GSE28948: TMPRSS2-ERG, HDACs and EZH2 are involved in an AR-centric transcriptional circuitry that calibrates androgenic response for prostate cancer progression (gene expression data) GSE28950: TMPRSS2-ERG, HDACs and EZH2 are involved in ...

ORGANISM(S): Homo sapiens

2012-04-26 | E-GEOD-28951 | biostudies-arrayexpress

NKX3-1, a Novel Transcriptional Factor of AR, Promotes Prostate Cancer Cell Survival via RAB3B GTPase-mediated protein trafficking (mRNA)

ORGANISM(S): Homo sapiens

2011-11-16 | E-GEOD-28596 | biostudies-arrayexpress

Androgen Receptor Variant AR-V9 Is Coexpressed with AR-V7 in Prostate Cancer Metastases and Predicts Abiraterone Resistance.

Not available

S-EPMC5644285 | biostudies-literature

RET Kinase Inhibition in Neuroendocrine Prostate Cancer, part 1

Increased treatment of metastatic castration resistant prostate cancer (mCRPC) with second-generation anti-androgen therapies (ADT) has coincided with a greater incidence of lethal, aggressive variant prostate cancer (AVPC) tumors that have lost androgen receptor (AR) signaling. AVPC tumors may also...

ORGANISM(S): Homo sapiens (Human)

2020-05-29 | PXD012970 | Pride

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