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Transcriptomics5-hydroxymethylcytosine marks promoters in colon that resist hypermethylation in cancer [ExpressionArray]
The discovery of cytosine hydroxymethylation (5-hmC) as a mechanism that potentially controls DNA methylation changes typical of neoplasia prompted us to investigate its behavior in colon cancer. 5-hmC is globally reduced in proliferating cells such as colon tumors and the gut crypt progenitors, fro...
ORGANISM(S): Homo sapiens 
2015-02-03 | E-GEOD-47063 | biostudies-arrayexpress
Genomics5-hydroxymethylcytosine marks promoters in colon that resist hypermethylation in cancer [seq]
The discovery of cytosine hydroxymethylation (5-hmC) as a mechanism that potentially controls DNA methylation changes typical of neoplasia prompted us to investigate its behavior in colon cancer. 5-hmC is globally reduced in proliferating cells such as colon tumors and the gut crypt progenitors, fro...
ORGANISM(S): Homo sapiens 
2015-02-03 | E-GEOD-47590 | biostudies-arrayexpress
GenomicsPol2Chip
Pol2
EGAD00010000860 | EGA
GenomicsH3K27me3Chip
H3K27me3
EGAD00010000862 | EGA
Genomicsena-DATASET-CRUK-CI-11-11-2015-15:28:33:473-12 - samples
Data from the paper Context-specific Effects of TGFβ/SMAD3 in Cancer Are Modulated by the Epigenome. Tufegdzic et al, Cell Reports 2015
EGAD00001001668 | EGA
GenomicsExpression
Expression Arrays
EGAD00010000867 | EGA
GenomicsH3K27AcChip
H3K27Ac
EGAD00010000863 | EGA
GenomicsMBDSEQ
MBDSEQ
EGAD00010000865 | EGA
Genomicsena-DATASET-CRUK-CI-11-11-2015-14:57:45:115-9 - samples
Data from the paper Context-specific Effects of TGFβ/SMAD3 in Cancer Are Modulated by the Epigenome. Tufegdzic et al, Cell Reports 2015
EGAD00001001667 | EGA
GenomicsAza
Targeted bisulfite sequencing
EGAD00010000868 | EGA
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