Dataset Information

Detecting Regions At Risk for Spreading COVID-19 Using Existing Cellular Wireless Network Functionalities.

ABSTRACT: Goal: The purpose of this article is to introduce a new strategy to identify areas with high human density and mobility, which are at risk for spreading COVID-19. Crowded regions with actively moving people (called at-risk regions) are susceptible to spreading the disease, especially if they contain asymptomatic infected people together with healthy people. Methods: Our scheme identifies at-risk regions using existing cellular network functionalities-handover and cell (re)selection-used to maintain seamless coverage for mobile end-user equipment (UE). The frequency of handover and cell (re)selection events is highly reflective of the density of mobile people in the area because virtually everyone carries UEs. Results: These measurements, which are accumulated over very many UEs, allow us to identify the at-risk regions without compromising the privacy and anonymity of individuals. Conclusions: The inferred at-risk regions can then be subjected to further monitoring and risk mitigation.

SUBMITTER: Alsaeedy AAR

PROVIDER: S-EPMC8043492 | biostudies-literature | 2020

REPOSITORIES: biostudies-literature

ACCESS DATA

Publications

Detecting Regions At Risk for Spreading COVID-19 Using Existing Cellular Wireless Network Functionalities.

Alsaeedy Alaa A R AAR Chong Edwin K P EKP

IEEE open journal of engineering in medicine and biology 20200615

Goal: The purpose of this article is to introduce a new strategy to identify areas with high human density and mobility, which are at risk for spreading COVID-19. Crowded regions with actively moving people (called at-risk regions) are susceptible to spreading the disease, especially if they contain asymptomatic infected people together with healthy people. Methods: Our scheme identifies at-risk regions using existing cellular network functionalities-handover a ...[more]

PMID: 34192281

Similar Datasets

Project description:Large, naturally evolved biomolecular networks typically fulfil multiple functions. When modelling or redesigning such systems, functional subsystems are often analysed independently first, before subsequent integration into larger-scale computational models. In the design and analysis process, it is therefore important to quantitatively analyse and predict the dynamics of the interactions between integrated subsystems; in particular, how the incremental effect of integrating a subsystem into a network depends on the existing dynamics of that network. In this paper we present a framework for simulating the contribution of any given functional subsystem when integrated together with one or more other subsystems. This is achieved through a cascaded layering of a network into functional subsystems, where each layer is defined by an appropriate subset of the reactions. We exploit symmetries in our formulation to exhaustively quantify each subsystem's incremental effects with minimal computational effort. When combining subsystems, their isolated behaviour may be amplified, attenuated, or be subject to more complicated effects. We propose the concept of mutual dynamics to quantify such nonlinear phenomena, thereby defining the incompatibility and cooperativity between all pairs of subsystems when integrated into any larger network. We exemplify our theoretical framework by analysing diverse behaviours in three dynamic models of signalling and metabolic pathways: the effect of crosstalk mechanisms on the dynamics of parallel signal transduction pathways; reciprocal side-effects between several integral feedback mechanisms and the subsystems they stabilise; and consequences of nonlinear interactions between elementary flux modes in glycolysis for metabolic engineering strategies. Our analysis shows that it is not sufficient to just specify subsystems and analyse their pairwise interactions; the environment in which the interaction takes place must also be explicitly defined. Our framework provides a natural representation of nonlinear interaction phenomena, and will therefore be an important tool for modelling large-scale evolved or synthetic biomolecular networks.

Project description:Background and objective Many countries around the world experienced a high increase in the number of COVID-19 cases after a few weeks of the first case, and along with it, excessive pressure on the healthcare systems. While medicines, drugs, and vaccines against the COVID-19 are being developed, social isolation has become the most used method for controlling the virus spreading. With the social isolation, authorities aimed to slow down the spreading, avoiding saturation of the healthcare system, and allowing that all critical COVID-19 cases could be appropriately treated. By tuning the proposed model to fit Brazil's initial COVID-19 data, the objectives of the paper are to analyze the impact of the social isolation features on the population dynamics; simulate the number of deaths due to COVID-19 and due to the lack of healthcare infrastructure; study combinations of the features for the healthcare system does not collapse; and analyze healthcare system responses for the crisis. Methods In this paper, a Susceptible-Exposed-Infected-Removed model is described in terms of probabilistic cellular automata and ordinary differential equations for the transmission of COVID-19, flexible enough for simulating different scenarios of social isolation according to the following features: the start day for the social isolation after the first death, the period for the social isolation campaign, and the percentage of the population committed to the campaign. Results Results showed that efforts in the social isolation campaign must be concentrated both on the isolation percentage and campaign duration to delay the healthcare system failure. For the hospital situation in Brazil at the beginning of the pandemic outbreak, a rate of 200 purchases per day of intensive care units and mechanical ventilators is the minimum rate to prevent the collapse of the healthcare system. Conclusions By using the model for different scenarios, it is possible to estimate the impact of social isolation campaign adhesion. For instance, if the social isolation percentage increased from 40% to 50% in Brazil, the purchase rate of 150 intensive care units and mechanical ventilators per day would be enough to prevent the healthcare system to collapse. Moreover, results showed that a premature relaxation of the social isolation campaign can lead to subsequent waves of contamination.

Dataset Information

Detecting Regions At Risk for Spreading COVID-19 Using Existing Cellular Wireless Network Functionalities.

Publications

Detecting Regions At Risk for Spreading COVID-19 Using Existing Cellular Wireless Network Functionalities.

Similar Datasets

OmicsDI is part of the ELIXIR infrastructure

Tweets