Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

UV-RIP experiments of ZNFX1 in VSV infected A549 cells

ABSTRACT: UV-RIP experiments of ZNFX1 in VSV infected A549 cells

PROVIDER: PRJNA549612 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Similar Datasets

UV-RIP experiments of ZNFX1 in VSV infected A549 cells

Project description:UV-RIP-seq was used to identify VSV-RNA associated with endogenous ZNFX1 in VSV infected A459 cells.

2019-08-30 | GSE132977 | GEO

UV cross-linking RIP-seq of VSV-GFP-infected HEK293T cells overexpressed PTENα

Project description:To determine whether PTENα directly binds to viral RNA or host mRNA, we performed UV cross-linking RNA immunoprecipitation (RIP) sequencing of HEK293T cells transfected with vector encoding PTENα under VSV-GFP (green fluorescent protein-expressing vesicular stomatitis virus) infection

2024-02-19 | GSE218439 | GEO

RIP- seq identified the interacted RNAs of SAFA with or without VSV infection

Project description:THP-1 cells were infected with VSV for 6 hours or 24 hours, RiP-seq was did with SAFA antibody.

2022-03-31 | GSE199677 | GEO

PDAP1 RIP in UV-crosslinked human T cells

Project description:PDAP1 RIP in UV-crosslinked human T cells

| PRJNA761680 | ENA

To survey ncRNAs that associate with Lsd1 or Bre1 complexes through RIP experiments

Project description:These paired RIP experiments were designed to determine specific ncRNAs that associated with either Lsd1 (Lysine specific demethylase 1) complex or Bre1.

2024-05-01 | GSE244906 | GEO

SILAC Experiment for A549 cells treated with UV and 2-deoxyglucose

Project description:SILAC labelled peptides from control and UV or 2-deoxy glucose treated A549 cells. Control is light labelled and treatment groups are heavy labelled. Fractions are as per below: UV1: APM-0331-2 to APM-0331-6 UV2: APM-0331-12 to APM-0331-16 2DG1: APM-0331-7 to APM-0331-11 2DG2: APM-0331-17 to APM-0331-21

2023-03-16 | MSV000091503 | MassIVE

RIp-chip experiments with proteins that do not contain RNA-binding domains

Project description:In order to assess the prevalence of cotranslational assembly of protein complexes we performed RIp-chip experiments with many proteins that do not conatin RNA-binding motifs

2012-05-20 | E-TABM-1158 | biostudies-arrayexpress

RIP- seq identified the interacted RNAs of SAFA with or without VSV infection

Project description:RIP- seq identified the interacted RNAs of SAFA with or without VSV infection

| PRJNA821177 | ENA

Analysis of global gene expression profiles of hPAF1 deficient A549 cells

Project description:This SuperSeries is composed of the following subset Series: GSE35265: Analysis of global gene expression profiles of hPAF1 deficiency on unstimulated A549 cells GSE35266: Analysis of global gene expression profiles of hPAF1 deficient A549 cells during infection with H1N1 influenza A virus or vesicular stomatitis virus (VSV) GSE35267: Analysis of global gene expression profiles of hPAF1 deficient A549 cells during stimulation with PR8/?NS1 influenza virus, IFN?1 or Poly(I:C) Refer to individual Series

2012-02-15 | E-GEOD-35268 | biostudies-arrayexpress

Analysis of global gene expression profiles of hPAF1 deficient A549 cells during infection with H1N1 influenza A virus or vesicular stomatitis virus (VSV)

Project description:Viral infection is commonly associated with virus-driven hijacking of host proteins. We describe a novel mechanism by which influenza virus impacts host cells through the interaction of influenza NS1 protein with the infected cell epigenome. We show that the NS1 protein of influenza A H3N2 targets the transcription elongation PAF1 complex (hPAF1C). We demonstrate that binding of NS1 to hPAF1C results in suppression of hPAF1C-mediated transcriptional elongation. More importantly,in the following data sets, we show that hPAF1 plays a crucial role in the antiviral response. Loss of hPAF1C reduces antiviral gene expression and reduces inducible transcription of target genes after stimulation with viral RNA analogue poly(I:C), vesicular stomatitis virus (VSV), exogenous recombinant IFN(beta) and influenza virus (H1N1). This study underscores the importance of hPAF1C in controlling inducible antiviral gene expression. Untreated (no siRNA), control siRNA-treated and hPAF1 siRNA-treated A549 cells were stimulated with A/Puerto Rico/8/1934 influenza virus (H1N1) or vesicular stomatitis virus (VSV). Total RNA was isolated with the Qiagen RNeasy mini kit. 200ng of total RNA per sample was used to prepare biotin-labeled RNA using MessageAmp™ Premier RNA Amplification Kit (Applied Biosystems) and hybridized to HumanHT-12 v4 Expression BeadChips (Illumina). Data analysis was performed using the GeneSpring GX11.0 software (Agilent Technologies). 3 biological replicates per condition

2012-02-15 | E-GEOD-35266 | biostudies-arrayexpress

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data