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ProteomicsCardiac Extracellular Matrix Proteomics of Ang II treated mice.
MS analysis of samples from left ventricles of mice treated with Ang II infusion for 2 weeks.
ORGANISM(S): Mus musculus (Mouse) 
2021-12-15 | PXD028887 | Pride
Heart Mouse. Extracellular matrix Angiotensin-2 
UnknownScoparone alleviates Ang II-induced pathological myocardial hypertrophy in mice by inhibiting oxidative stress.
Not available
S-EPMC7957216 | biostudies-literature
UnknownAssociation between circulating levels of ACE2-Ang-(1-7)-MAS axis and ACE2 gene polymorphisms in hypertensive patients.
Not available
S-EPMC4998461 | biostudies-literature
UnknownOsthole protects against Ang II-induced endotheliocyte death by targeting NF-κB pathway and Keap-1/Nrf2 pathway.
Not available
S-EPMC6357313 | biostudies-literature
UnknownGJA1-20k attenuates Ang II-induced pathological cardiac hypertrophy by regulating gap junction formation and mitochondrial function.
Not available
S-EPMC8115281 | biostudies-literature
UnknownExogenous Ang-(1-7) inhibits autophagy via HIF-1α/THBS1/BECN1 axis to alleviate chronic intermittent hypoxia-enhanced airway remodelling of asthma.
Not available
S-EPMC10545676 | biostudies-literature
UnknownAng II Controls the Expression of Mapkap1 by miR-375 and Affects the Function of Islet β Cells.
Not available
S-EPMC10514520 | biostudies-literature
UnknownIrisin attenuates vascular remodeling in hypertensive mice induced by Ang II by suppressing Ca2+-dependent endoplasmic reticulum stress in VSMCs.
Not available
S-EPMC10758105 | biostudies-literature
TranscriptomicsLIN28 eRIP in Differentiating hESCs
Excluded-volume-effect-optimized RNA-IP (eRIP) of LIN28-associated mRNAs in H1 hESCs. H1s are induced to differentiate into either trophoblast (TE) or neural (N) lineages, and LIN28 eRIP performed at 0hr (Undifferentiated), 12hr TE, 24hr TE and 24hr N
ORGANISM(S): Homo sapiens 
2014-07-21 | E-MTAB-1652 | biostudies-arrayexpress
TranscriptomicsGene expression profiling in cardiac AngII-overexpressing mice (TG1306/1R)
Agonist-induced cardiac hypertrophy in TG1306/1R transgenic mice with cardiac angiotensin II overproduction leads to a gradual transition from a compensatory hypertrophic state to heart failure. To gain insight into the molecular mechanisms that play a role in maintaining cardiac integrity in the di...
ORGANISM(S): Mus musculus 
2010-05-18 | E-GEOD-13336 | biostudies-arrayexpress
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