Bcl-w - OmicsDI

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These mice have a relatively normal first wave of spermatogenesis but never become fertile. Older animals show increased apoptosis of germ cells, increasing in severity as germ cell stage advances. (1)
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Tags

xref:PubMed:21893020 (1)
xref:PubMed:17234957 (1)
xref:PubMed:27805565 (1)
xref:PubMed:38521953 (1)
xref:PubMed:18451170 (1)

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Exosomal miRNA profiling in Bcl-w overexpressed U251cells

PRJNA1083993 | ENA

Inhibition of Oligomeric BAX by an Anti-Apoptotic Dimer

BAX is a pro-apoptotic BCL-2 protein that resides in the cytosol as a monomer until triggered by cellular stress to transform into an oligomer that permeabilizes the mitochondria and induces apoptosis. We previously reported the generation of a full-length BAX oligomer (BAXO) that recapitulates pro-...

ORGANISM(S): Homo sapiens (Human)

2025-11-21 | PXD049315 | Pride

Bcl-w protects hippocampus during experimental status epilepticus.

Not available

S-EPMC1988875 | biostudies-literature

Transcription profiling of mouse models of sepsis cecal ligation and puncture and tracheal instillation of P. aeruginosa reveals bcl-2 overexpression modulates transcription responses in vivo

Background: We hypothesized that spleen microarray gene expression profiles analyzed with contemporary pathway analysis software would provide molecular pathways of interest and target genes that might help explain the affect of bcl-2 on improving survival during sepsis. Methods: Two mouse models of...

ORGANISM(S): Mus musculus

2008-06-14 | E-GEOD-5811 | biostudies-arrayexpress

Transcription profiling of human SCLC cell line set 2

RNA expression analysis was performed to compare patterns to sensitivity to BCL2 inhibitors (ABT-263). Overexpression of the prosurvival Bcl-2 family members (Bcl-2, Bcl-xL and Mcl-1) is commonly associated with tumor maintenance, progression and chemoresistance. We previously reported the discover...

ORGANISM(S): Homo sapiens

2008-06-19 | E-GEOD-10841 | biostudies-arrayexpress

BCL-W has a fundamental role in B cell survival and lymphomagenesis.

Not available

S-EPMC5272186 | biostudies-literature

Directed elimination of senescent cells by inhibition of BCL-W and BCL-XL.

Not available

S-EPMC4823827 | biostudies-literature

BCL-W makes only minor contributions to MYC-driven lymphoma development.

Not available

S-EPMC10491490 | biostudies-literature

BCL-W is a regulator of microtubule inhibitor-induced mitotic cell death.