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UnknownExpression and disruption of the Arabidopsis TOR (target of rapamycin) gene.
Not available
S-EPMC122964 | biostudies-literature
no omics definedTranscription profiling of S. cerevisiae strains with controlled expression of hmo1 and Ifh1
Effect of hmo1 on yeast transcriptional response, as compared to Ifh1
ORGANISM(S): Saccharomyces cerevisiae 
2007-09-17 | E-MEXP-651 | biostudies-arrayexpress
TranscriptomicsSenescence leads to epigenomic disruption invoking hallmarks of reprogramming and pluripotency
This SuperSeries is composed of the SubSeries listed below. Refer to individual Series
ORGANISM(S): Homo sapiens 
2014-12-15 | E-GEOD-53332 | biostudies-arrayexpress
ProteomicsSequential digestion with Trypsin and Elastase in cross-linking/mass spectrometry
Cross-linking/mass spectrometry has become an important approach for studying protein structures and protein-protein interactions. The amino acid composition of some protein regions impedes the detection of cross-linked residues, although it would yield invaluable information for protein modelling. ...
ORGANISM(S): Homo sapiens (Human) Schizosaccharomyces pombe 
2019-03-07 | PXD011459 | Pride
Lc-msms Cross-linking Yeast Human taf4-12 complex 
UnknownB cell-derived acetylcholine mitigates skin inflammation in mice through α9 nicotinic acetylcholine receptor-mediated signaling.
Not available
S-EPMC12054817 | biostudies-literature
GenomicsLamin B1 ChIPseq experiments from ER:RAS IMR90s
Lamin B1 from OIS IMR90s We used ChIP-seq to examine the global binding of Lamin B1 in human IMR90 oncogene-induced senescence
ORGANISM(S): Homo sapiens 
2014-12-15 | E-GEOD-53331 | biostudies-arrayexpress
GenomicsH3K9me3 ChIPseq experiments from PD IMR90s
H3K9me3 ChIPseq in Proliferating and Senescent IMR90s We used ChIP-seq to examine the global binding of H3K9me3 in human IMR90 replicative senescence (RS) and oncogene-induced (OIS)
ORGANISM(S): Homo sapiens 
2014-12-15 | E-GEOD-53329 | biostudies-arrayexpress
UnknownInflammasome Signaling and Impaired Vascular Health in Psoriasis.
Not available
S-EPMC6436998 | biostudies-literature
GenomicsMutational analysis reveals the origin and therapy-driven evolution of recurrent glioma
Tumor recurrence is a leading cause of cancer mortality. Therapies for recurrent disease may fail, at least in part, because the genomic alterations driving the growth of recurrences are distinct from those in the initial tumor. To explore this hypothesis, we sequenced the exomes of 23 initial low-g...
EGAS00001000579 | EGA
GenomicsEvolution of DNA repair defects during malignant progression of low-grade gliomas after temozolomide treatment.
Temozolomide (TMZ) increases the overall survival of patients with glioblastoma (GBM), but its role in the clinical management of diffuse low-grade gliomas (LGG) is still being defined. DNA hypermethylation of the O 6 -methylguanine-DNA methyltransferase (MGMT) promoter is associated with an improve...
EGAS00001001179 | EGA
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