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GenomicsSMAD2/3 chromatin immunoprecipitation sequencing (ChIP-seq) in human naïve and primed human pluripotent stem cells
The aim of this experiment was to investigate the role of TGFβ signalling pathway in human pluripotency, through ChIP-seq analysis of its main downstream effector SMAD2/3 in naïve and primed human pluripotent stem cells (hPSCs).
ORGANISM(S): Homo sapiens 
2021-08-13 | E-MTAB-10017 | biostudies-arrayexpress
TranscriptomicsSingle cell RNA sequencing of naive and primed human pluripotent stem cells treated with SB431542
The aim of this experiment was to investigate the role of TGFβ signalling in human pluripotency, looking at the effect of SB431542 (SB), a potent and selective SMAD inhibitor that blocks TGFβ/Activin receptors ALK5, ALK4, and ALK7, on gene expression. We performed 10X sequencing in naïve and primed ...
ORGANISM(S): Homo sapiens 
2021-08-13 | E-MTAB-10018 | biostudies-arrayexpress
MetabolomicsSerum-based metabolomics characterization of pigs treated with ractopamine
A study was performed to determine if serum-metabolomics could be used to establish a predictive tool for identifying ractopamine misuse in pigs. Ractopamine, a beta-agonist used as growth promoter in livestock, is with great controversy, and it has been forbidden in most countries worldwide. Howeve...
2017-06-12 | MTBLS384 | MetaboLights
UnknownGWAS of multiple neuropathology endophenotypes identifies new risk loci and provides insights into the genetic risk of dementia.
Not available
S-EPMC11549054 | biostudies-literature
UnknownAuthor Correction: GWAS of multiple neuropathology endophenotypes identifies new risk loci and provides insights into the genetic risk of dementia.
Not available
S-EPMC12283389 | biostudies-literature
UnknownPharmacological plus optical penalization treatment for amblyopia: results of a randomized trial.
Not available
S-EPMC2713107 | biostudies-literature
UnknownStereoacuity in children with anisometropic amblyopia.
Not available
S-EPMC3223370 | biostudies-literature
UnknownAccelerated functional brain aging in pre-clinical familial Alzheimer's disease.
Not available
S-EPMC8429427 | biostudies-literature
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