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ProteomicsNucleolin is a cell surface receptor of C1QTNF4
C1QTNF4 is a secreted protein that is structurally unique within the C1QTNF family, a family of proteins that have structural homology with both complement C1q and the tumor necrosis factor superfamily. The role of C1QTNF4 in immunity and inflammation remains poorly defined and a cell surface recept...
ORGANISM(S): Homo sapiens (Human) 
2021-03-21 | PXD023088 | Pride
Innate immunity Nucleolin Cell surface receptor Inflammation Ctrp4 C1qtnf4 Monocyte Autoimmune disease Systemic lupus erythematosus 
UnknownNucleolin acts as the receptor for C1QTNF4 and supports C1QTNF4-mediated innate immunity modulation.
Not available
S-EPMC8042453 | biostudies-literature
UnknownC1q/TNF-related protein 4 mediates proliferation and migration of vascular smooth muscle cells during vascular remodelling.
Not available
S-EPMC10206021 | biostudies-literature
TranscriptomicsCRISPRi-targeted raw RNA-seq data in iPSC-derived neurons. In this study, we performed RNA-sequencing to test transcriptomic changes of CRISPRi-targetd MIR family TE (chr11: 47608036-47608220) in iPSC-derived neurons.
Despite its broad role in human disease, transposable element (TE) dysregulation of quantitative trait loci (QTL) has not been well-characterized in brain aging or Alzheimer’s disease (AD). Here, we leveraged large-scale human RNA-sequencing (RNA-seq), whole-genome sequencing (WGS), and QTL data (xQ...
ORGANISM(S): Homo sapiens 
2024-06-01 | GSE246052 | GEO
UnknownWidespread transposable element dysregulation in human aging brains with Alzheimer's disease.
Not available
S-EPMC11567813 | biostudies-literature
UnknownDissecting the shared genetic architecture between Alzheimer's disease and frailty: a cross-trait meta-analyses of genome-wide association studies.
Not available
S-EPMC11069310 | biostudies-literature
UnknownDe novo mutations implicate novel genes in systemic lupus erythematosus.
Not available
S-EPMC5886157 | biostudies-literature
GenomicsCRISPRi-targeted raw RNA-seq data in iPSC-derived neurons. In this study, we performed RNA-sequencing to test transcriptomic changes of CRISPRi-targetd MIR family TE (chr11: 47608036-47608220) in iPSC-derived neurons.
CRISPRi-targeted raw RNA-seq data in iPSC-derived neurons. In this study, we performed RNA-sequencing to test transcriptomic changes of CRISPRi-targetd MIR family TE (chr11: 47608036-47608220) in iPSC-derived neurons.
PRJNA1031180 | ENA
UnknownCTRP4 ablation impairs associative learning and memory.
Not available
S-EPMC9675051 | biostudies-literature
UnknownMulti-omics integration reveals a six-malignant cell maker gene signature for predicting prognosis in high-risk neuroblastoma.
Not available
S-EPMC9687102 | biostudies-literature
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