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GenomicsEffect of CAMK2D inhibition in RBM20 loss of function mouse models
Effect of CAMK2D inhibition in RBM20 loss of function mouse models
PRJNA1369419 | ENA
ProteomicsCAMK2D recruits RNF8-MAD2 complex to RNA splicing proteins for mitotic checkpoint activation in GBM
Glioblastoma (GBM) is a lethal brain tumor with remarkable intra-tumoral heterogeneity and therapeutic resistance which is linked to glioma stem cell (GSC). GSC contains unstable genome and elevated genomic instability could impair GSC self-renewal and tumorigenicity. In addition, GSC displays great...
ORGANISM(S): Homo Sapiens (human) 
2023-07-21 | PXD038123 | JPOST Repository
ProteomicsUsing CRISPR-Cas9/Phosphoproteomics to Identify Kinase Substrates: Calmodulin Kinase 2 Delta
The Ca2+/CaM-dependent protein kinase 2 (CAMK2) family of protein kinases are key protein kinases involved in regulation of cellular processes in a variety of tissues including brain, smooth muscle and kidney. One member, CAMK2-delta (CAMK2D), has been proposed to be involved in vasopressin signali...
ORGANISM(S): Mus musculus (Mouse) 
2023-11-06 | PXD040159 | Pride
Mass spectrometry Camk2d Vasopressin Aqp2 
UnknownRole of CAMK2D in neurodevelopment and associated conditions.
Not available
S-EPMC10870144 | biostudies-literature
TranscriptomicsEffect of CAMK2D inhibition in RBM20 loss of function mouse models
Mutations in RBM20, a splicing factor that targets multiple pivotal cardiac genes including TTN and CAMK2D, cause a clinically aggressive form of dilated cardiomyopathy (DCM) with a high risk of malignant ventricular arrhythmias. We hypothesized that the RBM20 target CAMK2D contributes critically to...
ORGANISM(S): Mus musculus 
2025-12-01 | GSE311410 | GEO
GenomicsTranscriptome changes in response to dDAVP stimulation in CRISPR/Cas9 mediated Camk2d knock-out mpkCCD cells
Transcriptome changes in response to dDAVP stimulation in CRISPR/Cas9 mediated Camk2d knock-out mpkCCD cells
PRJNA951435 | ENA
GenomicsCaMK2D is a critical regulator of coactivation of leukemia stem cell factors in acute myeloid leukemia.
CaMK2D is a critical regulator of coactivation of leukemia stem cell factors in acute myeloid leukemia.
PRJNA675223 | ENA
TranscriptomicsTranscriptome changes in response to dDAVP stimulation in CRISPR/Cas9 mediated Camk2d knock-out mpkCCD cells
The Ca2+/CaM-dependent protein kinase 2-delta (CAMK2D) has been proposed to be involved in vasopressin signaling in the renal collecting duct, which controls water and salt excretion by the kidney. RNA sequancing and quantitative proteomics analyses identified the expression of multiple CAMK2 isofor...
ORGANISM(S): Mus musculus 
2023-06-01 | GSE228762 | GEO
UnknownCAMK2D and Complement Factor I-Involved Calcium/Calmodulin Signaling Modulates Sodium Iodate-Induced Mouse Retinal Degeneration.
Not available
S-EPMC11781327 | biostudies-literature
TranscriptomicsCaMK2D is a critical regulator of coactivation of leukemia stem cell factors in acute myeloid leukemia.
Relapse is one of the biggest challenges in treating acute myeloid leukemia (AML) due to leukemia stem cells (LSCs) resisting chemotherapy, providing a cellular reservoir to form the basis for relapse. Here, we identify CaMK2D is a critical regulator of stemness and leukemogenesis of AML. Transcript...
ORGANISM(S): Homo sapiens 
2021-11-08 | GSE161032 | GEO
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