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ProteomicsMass spectrometry-based characterisation of human megakaryocyte CRLF3 interactome
Cytokine-receptor like factor 3 (CRLF3) is a protein with an as yet unknown function. CRLF3 has been shown to interact with the members of the Hippo pathway in HEK cells but the function of this interaction was not elucidated. Crlf3 null mice are viable and health but present with an isolated reduce...
ORGANISM(S): Homo sapiens (Human) 
2022-02-04 | PXD017026 | Pride
Human Crlf3 Megakaryocyte Lc-ms/ms 
UnknownCytokine Receptor-Like Factor 3 (CRLF3) Contributes to Early Zebrafish Hematopoiesis.
Not available
S-EPMC9251315 | biostudies-literature
UnknownThe cytokine receptor CRLF3 is a human neuroprotective EV-3 (Epo) receptor.
Not available
S-EPMC10165946 | biostudies-literature
UnknownThe Insect Ortholog of the Human Orphan Cytokine Receptor CRLF3 Is a Neuroprotective Erythropoietin Receptor.
Not available
S-EPMC5509957 | biostudies-literature
UnknownThe Orphan Cytokine Receptor CRLF3 Emerged With the Origin of the Nervous System and Is a Neuroprotective Erythropoietin Receptor in Locusts.
Not available
S-EPMC6797617 | biostudies-literature
UnknownMultiple Mammalian Cytokines and Erythropoietin-Mimetic Peptides Protect Insect Neurons via Phylogenetically Conserved Cytokine Receptor-Like Factor 3 (CRLF3).
Not available
S-EPMC12409108 | biostudies-literature
UnknownCRLF3 plays a key role in the final stage of platelet genesis and is a potential therapeutic target for thrombocythemia.
Not available
S-EPMC7614665 | biostudies-literature
GenomicsPatient-derived iPSC-cerebral organoid modeling of the 17q11.2 microdeletion syndrome establishes CRLF3 as a critical regulator of neurogenesis
Patient-derived iPSC-cerebral organoid modeling of the 17q11.2 microdeletion syndrome establishes CRLF3 as a critical regulator of neurogenesis
PRJNA699232 | ENA
TranscriptomicsPatient-derived iPSC-cerebral organoid modeling of the 17q11.2 microdeletion syndrome establishes CRLF3 as a critical regulator of neurogenesis
Neurodevelopmental disorders are often caused by chromosomal microdeletions encompassing numerous contiguous genes. One such microdeletion on chromosome 17q11.2, involving the NF1 gene and flanking regions ( NF1 total gene deletion; NF1 -TGD), occurs in a subset of Neurofibromatosis type 1 (NF1) pat...
ORGANISM(S): Homo sapiens 
2021-06-07 | GSE166080 | GEO
UnknownPatient-derived iPSC-cerebral organoid modeling of the 17q11.2 microdeletion syndrome establishes CRLF3 as a critical regulator of neurogenesis.
Not available
S-EPMC8278229 | biostudies-literature
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