Sort   by:  
 Page size 
TranscriptomicsLow Input Micro-C (Pico-C) data of Drosophila melanogaster embryos across early nuclear cycles
Low-input Micro-C (Pico-C) was performed on hand-sorted PCNA-eGFP embryos at interphase from nuclear cycles (NCs) 9 to 14, including double knockdowns of Zelda and GAF and their respective controls (NC14), as well as embryos injected with α-amanitin or water (NC14). ATAC-seq was performed on double ...
ORGANISM(S): Drosophila melanogaster 
2025-11-10 | E-MTAB-14477 | biostudies-arrayexpress
TranscriptomicsHost and pathogen interaction: time course [C. albicans]
The interaction between fungal pathogen and host during infection is a complex and dynamic process. To resolve this, we chose the zebrafish model organism as the host to study C. albicans infection via systems biology approach. Transcriptome microarray data and histological analysis of surviving fis...
ORGANISM(S): Candida albicans 
2013-08-26 | E-GEOD-32117 | biostudies-arrayexpress
TranscriptomicsMicro-C of Drosophila embryos to investigate chromatin conformation during dorsal-ventral patterning
Micro-C was carried out for control embryos and embryos produced from gd7 mutant mothers. The embryos from mutant mothers produce only a single type along the dorsal-ventral axis (dorsal ectoderm). We used these embryos to compare chromatin conformation across tissues.
ORGANISM(S): Drosophila melanogaster 
2021-01-08 | E-MTAB-9784 | biostudies-arrayexpress
TranscriptomicsDrosophila melanogaster Micro-C experiments of wild-type and ectopic E3 enhancer-inserted Drosophila embryos
Micro-C in Drosophila embryos from a wild-type line and from transgenic fly line twiΔE3, E3(+51kb) at 5-8 hrs after egg lay. Two independent collections were performed. Data from the two genotypes were analysed and compared to understand how the ectopic insertion of an enhancer affects global chroma...
ORGANISM(S): Drosophila melanogaster 
2023-08-01 | E-MTAB-12146 | biostudies-arrayexpress
MetabolomicsProfiling aromatic amino acid metabolome in the human gut microbiota reveals Clostridioides difficile specific N-acyl amino acids

Owing to its compositional and chemical complexity, much of the gut microbiota metabolome remains poorly characterized. Aromatic amino acids (AAAs) have a history of being privileged substrates for the biosynthesis of diverse bioactive metabolites and thus represent a potentially rich source of b...

2026-07-23 | MTBLS13404 | MetaboLights
TranscriptomicsHost and pathogen interaction: time course [zebrafish]
The interaction between fungal pathogen and host during infection is a complex and dynamic process. To resolve this, we chose the zebrafish model organism as the host to study C. albicans infection via systems biology approach. Transcriptome microarray data and histological analysis of surviving fis...
ORGANISM(S): Danio rerio 
2013-08-26 | E-GEOD-32118 | biostudies-arrayexpress
UnknownThe C-terminal region of G72 increases D-amino acid oxidase activity.
Not available
S-EPMC3907796 | biostudies-literature
TranscriptomicsMapping nucleosome resolution chromosome folding in yeast by Micro-C
We describe a Hi-C based method, Micro-C, in which micrococcal nuclease is used instead of restriction enzymes to fragment chromatin, enabling nucleosome resolution chromosome folding maps. Analysis of Micro-C maps for budding yeast reveals abundant self-associating domains similar to those reported...
ORGANISM(S): Saccharomyces cerevisiae 
2015-06-25 | E-GEOD-68016 | biostudies-arrayexpress
TranscriptomicsConserved immune regulator HLH-30/TFEB couples pore-forming toxin damage sensing to intestine homeostasis via TTC38 signal orchestrator
Effector-triggered immunity (ETI) safeguards intestinal epithelial cells (IECs) by surveilling bacterial virulence effectors and effector-induced cellular stress, such as plasma membrane damage induced by pore-forming toxins (PFTs). However, the knowledge about ETI regulators in maintaining IEC home...
ORGANISM(S): Caenorhabditis elegans 
2025-11-11 | E-MTAB-15698 | biostudies-arrayexpress
UnknownImpact of interleukin-18 polymorphisms -607A/C and -137G/C on oral cancer occurrence and clinical progression.
Not available
S-EPMC3862691 | biostudies-literature
Sort   by:  
 Page size