Jamnick N A NA - OmicsDI

Show results for

Proteomics (2)
Unknown (1)

Organisms

Alphavirus (2)
Anemia (2)
Archaea (2)
Bacteria (2)
Betaherpesvirinae (2)
Borrelia (2)
Chlorella vulgaris (2)
Clostridium perfringens (2)
Dengue virus (2)
Enterovirus A71 (2)
Eukaryota (2)
Hepatitis B virus (2)
Human alphaherpesvirus 1 (2)
Human betaherpesvirus 5 (2)
Influenza A virus (2)
Influenza C virus (2)
Japanese encephalitis virus group (2)
Kamptonema sp. (2)
Listeria monocytogenes (2)
Mammarenavirus (2)
Miscanthus x giganteus (2)
Mycobacterium tuberculosis (2)
Orthoflavivirus (2)
Panicum virgatum (2)
Pseudomonas aeruginosa (2)
Rattus (2)
Severe acute respiratory syndrome coronavirus 2 (2)
Staphylococcus aureus (2)
Streptococcus pneumoniae (2)
Viruses (2)

Repository

pride (2)

Tissue

Skeletal muscle (2)

Technology Type

Mass Spectrometry (2)
Data-independent acquisition (1)
Shotgun proteomics (1)

Publication Date

2026 (1)
2021 (1)

Release Date

2025 (1)

Lab affiliation

Department of Biochemistry and Pharmacology, Bio21 Molecular Science and Biotechnology Institute, The University of Melbourne, Melbourne, VIC, 3010, Australia 2Murdoch Children’s Research Institute, Melbourne, VIC 3052, Australia 3Victorian Clinical Genetics Services, Murdoch Children’s Research Institute, Melbourne, VIC, 3052, Australia (1)
University of Melbourne (1)

Previous page
1 / 1
You're on page 1
Next page

Sort by:

Relevance

Page size

Sprint interval exercise disrupts mitochondrial ultrastructure driving a unique mitochondrial stress response and remodelling in men.

Not available

S-EPMC12769668 | biostudies-literature

Training-induced bioenergetic improvement in human skeletal muscle is associated with non-stoichiometric changes in the mitochondrial proteome without reorganisation of respiratory chain content

Mitochondrial defects are implicated in multiple diseases and aging. Exercise training is an accessible and inexpensive therapeutic intervention that can improve mitochondrial bioenergetics and quality of life. By combining a multi-omics approach with biochemical and in silico normalisation, we remo...

ORGANISM(S): Homo sapiens (Human)

2021-09-30 | PXD026219 | Pride

Mitochondrial stress drives skeletal muscle remodelling following sprint-interval exercise.

Mitochondria play a central role in cellular metabolism and are crucial for maintaining cellular health. Exercise is a proven intervention to enhance mitochondrial function and prevent non-communicable diseases, yet the molecular mechanisms driving exercise-induced mitochondrial remodelling remain u...