Sort   by:  
 Page size 
GenomicsComparison of normal and KCNQ3 overexpressing HEK293T cells
Comparison of normal and KCNQ3 overexpressing HEK293T cells
PRJNA640046 | ENA
ProteomicsKCNQ2 and KCNQ5 can form a channel complex in the brain independent of KCNQ3 channels
KCNQ2 and KCNQ3 channels are associated with multiple neurodevelopmental disorders and are also therapeutic targets for neurological and neuropsychiatric diseases. The current dogma is that, in the brain, KCNQ2 forms diheteromeric channels with KCNQ3, but not with KCNQ5, a channel also resident in n...
ORGANISM(S): Homo sapiens (Human) 
2022-03-24 | PXD028142 | Pride
Kcnq5 Kcnq3 Epilepsy Autism spectrum disorders Co-immunoprecipitation Mass spectrometry Kcnq2 Neurodevelopmental disorders Channelopathy 
TranscriptomicsComparison of normal and KCNQ3 overexpressing HEK293T cells
KCNQ3 (KQT-like potassium channel member 3) was overexpressed in HEK293T cells and compared to control
ORGANISM(S): Homo sapiens 
2022-06-01 | GSE152673 | GEO
UnknownPore determinants of KCNQ3 K+ current expression.
Not available
S-EPMC3368142 | biostudies-literature
TranscriptomicsPan-cancer analysis reveals oncogenic roles of KCNQ3 in tumor progression, immune microenvironment remodeling, and its mechanistic dependence on ERK1/2 signaling in cholangiocarcinoma
Background: Potassium voltage-gated channel subfamily Q member 3 (KCNQ3) is primarily recognized for its function in neuronal excitability; however, its expression patterns, genetic landscape, and functional significance across human malignancies remain largely unexplored. This study aimed to system...
ORGANISM(S): Homo sapiens 
2026-06-30 | GSE331276 | GEO
UnknownKCNQ2 and KCNQ5 form heteromeric channels independent of KCNQ3.
Not available
S-EPMC9060518 | biostudies-literature
UnknownIncreased KCNQ3 expression in papillary thyroid cancer promotes proliferation and migration.
Not available
S-EPMC12625505 | biostudies-literature
UnknownLipophilic compounds restore function to neurodevelopmental-associated KCNQ3 mutations.
Not available
S-EPMC11413209 | biostudies-literature
UnknownBehavioral and neuro-functional consequences of eliminating the KCNQ3 GABA binding site in mice.
Not available
S-EPMC10248464 | biostudies-literature
UnknownPore helix-S6 interactions are critical in governing current amplitudes of KCNQ3 K+ channels.
Not available
S-EPMC3368123 | biostudies-literature
Sort   by:  
 Page size