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GenomicsGenome wide association study of leprosy in Chinese population

Leprosy is a chronic infectious disease caused by Mycobacterium leprae (M. leprae). Due to M. leprae's narrow host range and an inability to be cultured in vitro, the biological investigation of this disease has been difficult. Host genetic factors have been ...

phs000217.v1.p1 | EGA
UnknownBeyond the Antagonism: Self-Labeled Xanthone Inhibitors as Modeled "Two-in-One" Drugs in Cancer Therapy.
Not available
S-EPMC6044579 | biostudies-literature
UnknownPhosphatase CDC25B Inhibitors Produced by Basic Alumina-Supported One-Pot Gram-Scale Synthesis of Fluorinated 2-Alkylthio-4-aminoquinazolines Using Microwave Irradiation.
Not available
S-EPMC6130795 | biostudies-literature
UnknownStretchable electrode enabled electrochemical mass spectrometry for <i>in situ</i> and complementary analysis of cellular mechanotransduction.
Not available
S-EPMC12171969 | biostudies-literature
UnknownCryo-EM structures of human GPR34 enable the identification of selective antagonists.
Not available
S-EPMC10523607 | biostudies-literature
UnknownSuppression of CCDC6 sensitizes tumor to oncolytic virus M1.
Not available
S-EPMC7749300 | biostudies-literature
MetabolomicsThe trans-omics landscape of COVID-19
The outbreak of coronavirus disease 2019 (COVID-19) is a global health emergency. Various omics results have been reported for COVID-19, but the molecular hallmarks of COVID-19, especially in those patients without comorbidities, have not been fully investigated. Here we collect blood samples from 2...
2021-10-06 | MTBLS2542 | MetaboLights
UnknownUpregulated astrocytic HDAC7 induces depression-like disorders via deacetylating PINK1 and inhibiting mitophagy.
Not available
S-EPMC12642251 | biostudies-literature
UnknownKruppel-like factor 12 regulates aging ovarian granulosa cell apoptosis by repressing SPHK1 transcription and sphingosine-1-phosphate (S1P) production.
Not available
S-EPMC10463260 | biostudies-literature
UnknownH2AX facilitates classical non-homologous end joining at the expense of limited nucleotide loss at repair junctions.
Not available
S-EPMC5737864 | biostudies-literature
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