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TranscriptomicsInfluence of ATM-mediated DNA damage response on genomic variation in human induced pluripotent stem cells (NimbleGen CNV)
Genome instability is a potential limitation to the research and therapeutic application of induced pluripotent stem cells (iPSCs). Observed genomic variations reflect the combined activities of DNA damage, cellular DNA damage response (DDR), and selection pressure in culture. To understand the cont...
ORGANISM(S): Homo sapiens 
2016-03-29 | E-GEOD-78717 | biostudies-arrayexpress
TranscriptomicsInfluence of ATM-mediated DNA damage response on genomic variation in human induced pluripotent stem cells (Affymetrix expression)
Genome instability is a potential limitation to the research and therapeutic application of induced pluripotent stem cells (iPSCs). Observed genomic variations reflect the combined activities of DNA damage, cellular DNA damage response (DDR), and selection pressure in culture. To understand the cont...
ORGANISM(S): Homo sapiens 
2016-03-29 | E-GEOD-78716 | biostudies-arrayexpress
TranscriptomicsInfluence of ATM-mediated DNA damage response on genomic variation in human induced pluripotent stem cells (Affymetrix CNV)
Genome instability is a potential limitation to the research and therapeutic application of induced pluripotent stem cells (iPSCs). Observed genomic variations reflect the combined activities of DNA damage, cellular DNA damage response (DDR), and selection pressure in culture. To understand the cont...
ORGANISM(S): Homo sapiens 
2016-03-29 | E-GEOD-78715 | biostudies-arrayexpress
UnknownChallenges in the size analysis of a silica nanoparticle mixture as candidate certified reference material.
Not available
S-EPMC4917587 | biostudies-literature
ProteomicsDetection of N‑glycoprotein associated with IgA nephropathy in urine as a potential diagnostic biomarker using glycosylated proteomic analysis
The present study aimed to clarify the potential diagnosis value of the urinary N-glycoproteins using mass spectrometry (MS) analysis in patients with IgA nephropathy (IgAN).
ORGANISM(S): Homo sapiens (Human) 
2023-08-13 | PXD041151 | Pride
Ms-based proteomics Urine Iga nephropathy Glycoproteins Biomarkers 
ProteomicsDIA quantitative protein mass spectrometry of RBE cells knockout NAT10
To further explore the role of NAT10 in ICC, we constructed stable NAT10-knockdown RBE cells using shRNA and performed DIA quantitative proteomic analysis to explore its influence on downstream protein fuction.
ORGANISM(S): Homo sapiens (Human) 
2026-05-25 | PXD058708 | Pride
Intrahepatic cholangiocarcinoma nat10 dia 
ProteomicsRice OsFIE2-FLAG IP-MS analysis
To analyze the interaction proteins of Polycomb complex PRC2 in rice, we used the FIE2-FLAG transgenic material to perform IP-MS analysis.
ORGANISM(S): Oryza Sativa 
PXD033370 |
ProteomicsMass spectrometry with DIA quantification of cerebrospinal fluid in 20 subarachnoid hemorrhage patients
We applied mass spectrometry with DIA quantification to identify biomarker candidates in CSF samples from a well-characterized cohort consisted of 20 SAH patients.
ORGANISM(S): Human Csf-associated Densovirus 
2022-03-08 | PXD032118 |
ProteomicsMyocardial Mitochondrial Antiviral Signaling Protein Promotes Heart Ischemia-reperfusion Injury via RIG-I Signaling-proteomic sequencing
Myocardial ischemia-reperfusion injury (MIRI) is a life-threatening complication of myocardial infarcts, with inner mitochondrial membrane protein dysfunction involved in MIRI-induced heart injury. The role of outer mitochondrial membrane protein MAVS is unknown. Here we show that MAVS expression in...
ORGANISM(S): Mus Musculus 
2025-03-20 | PXD062097 |
ProteomicsMyocardial Mitochondrial Antiviral Signaling Protein Promotes Heart Ischemia-reperfusion Injury via RIG-I Signaling-mass spectrometry
Myocardial ischemia-reperfusion injury (MIRI) is a life-threatening complication of myocardial infarcts, with inner mitochondrial membrane protein dysfunction involved in MIRI-induced heart injury. The role of outer mitochondrial membrane protein MAVS is unknown. Here we show that MAVS expression in...
ORGANISM(S): Mus Musculus 
2025-03-20 | PXD062096 |
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