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MetabolomicsToxicometabolomics of the new psychoactive substances α-PBP and α-PEP studied in HepaRG cell incubates by means of untargeted metabolomics revealed unexpected amino acid adducts
Toxicometabolomics, essentially applying metabolomics to toxicology of endogenous compounds such as drugs of abuse or new psychoactive substances (NPS), can be investigated by using different in vitro models and dedicated metabolomics techniques to enhance the number of relevant findings. The presen...
2023-10-10 | MTBLS1481 | MetaboLights
UnknownStructure and function of H+/K+ pump mutants reveal Na+/K+ pump mechanisms.
Not available
S-EPMC9463140 | biostudies-literature
UnknownK(2)LaCl(5).
Not available
S-EPMC3011500 | biostudies-literature
UnknownResolving Complex K-Pt-Sn Interactions in PtSn@K-MFI Catalysts for Alkane Dehydrogenation.
Not available
S-EPMC12006991 | biostudies-literature
MetabolomicsLiquid Chromatography-High-Resolution Mass Spectrometry-Based In Vitro Toxicometabolomics of the Synthetic Cathinones 4-MPD and 4-MEAP in Pooled Human Liver Microsomes
Synthetic cathinones belong to the most often seized new psychoactive substances on an international level. This study investigated the toxicometabolomics, particularly the in vitro metabolism of 2-(methylamino)-1-(4-methylphenyl)-1-pentanone (4-MPD) and 2-(ethylamino)-1-(4-methylphenyl)-1-pentanone...
2023-11-28 | MTBLS2218 | MetaboLights
UnknownOn the effect of hyperaldosteronism-inducing mutations in Na/K pumps.
Not available
S-EPMC5677107 | biostudies-literature
UnknownNa/K Pump Mutations Associated with Primary Hyperaldosteronism Cause Loss of Function.
Not available
S-EPMC6779590 | biostudies-literature
MetabolomicsUntargeted metabolomics links glutathione to bacterial cell cycle progression
Cell cycle progression requires the coordination of cell growth, chromosome replication, and division. Consequently, a functional cell cycle must be coupled with metabolism. However, direct measurements of metabolome dynamics remained scarce, in particular in bacteria. Here, we describe an untargete...
2020-10-15 | MTBLS1402 | MetaboLights
GenomicsThe Growth factor independence 1 (GFI1) 36N variant predisposes to Acute Myeloid Leukemia (AML) by inducing epigenetic changes at the Hoxa9 locus
GFI136N is a coding Single Nucleotide Polymorphism (SNP) in the gene GFI1 that increases the risk for Acute myeloid leukemia (AML) by 60%. It is present in 3-5% of Caucasians and has a prevalence of 12% among AML patients. We generated knockin mice expressing either the human GFI136N variant or the ...
ORGANISM(S): Mus musculus 
2012-12-04 | E-GEOD-31657 | biostudies-arrayexpress
UnknownProton transfer in the K-channel analog of B-type Cytochrome c oxidase from Thermus thermophilus.
Not available
S-EPMC4223178 | biostudies-literature
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