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UnknownDecoding of methylated histone H3 tail by the Pygo-BCL9 Wnt signaling complex.
Not available
S-EPMC2726290 | biostudies-literature
UnknownEvolutionary adaptation of the fly Pygo PHD finger toward recognizing histone H3 tail methylated at arginine 2.
Not available
S-EPMC3851687 | biostudies-literature
UnknownStructural basis of the interaction between BCL9-Pygo and LDB-SSBP complexes in assembling the Wnt enhanceosome.
Not available
S-EPMC10287724 | biostudies-literature
GenomicsBcl9 and Pygo synergize downstream of Apc to effect intestinal neoplasia in mouse models recapitulating Familial Adenomatous Polyposis (Illumina Beadchip)
Bcl9 and Pygo synergize downstream of Apc to effect intestinal neoplasia in mouse models recapitulating Familial Adenomatous Polyposis (Illumina Beadchip)
PRJNA495815 | ENA
TranscriptomicsBcl9 and Pygo synergize downstream of Apc to effect intestinal neoplasia in mouse models recapitulating Familial Adenomatous Polyposis (Illumina Beadchip)
Bcl9 and Pygo function within the Wnt enhanceosome to effect β-catenin-dependent transcription. Whether they also mediate β-catenin-dependent neoplasia is unclear. Here we assess their roles in intestinal tumorigenesis initiated by Apc loss-of-function (ApcMin). Gene expression profiling revealed th...
ORGANISM(S): Mus musculus 
2018-12-31 | GSE121130 | GEO
UnknownMutations in Bcl9 and Pygo genes cause congenital heart defects by tissue-specific perturbation of Wnt/β-catenin signaling.
Not available
S-EPMC6217730 | biostudies-literature
UnknownCompetitive binding of a benzimidazole to the histone-binding pocket of the Pygo PHD finger.
Not available
S-EPMC4330097 | biostudies-literature
UnknownBcl9 and Pygo synergise downstream of Apc to effect intestinal neoplasia in FAP mouse models.
Not available
S-EPMC6374407 | biostudies-literature
ProteomicsPull down of full length and mutant BCL9 (deltaHD1) in mouse colorectal tumors and colonic epithelium
Bcl9 and Bcl9l (Bcl9l) are commonly thought as beta-catenin-dependent Wnt signaling transcriptional activators. Here we searched for BCL9 interacts that could explain the PYGO-independent phenotype induced be the loss of the HD1 (PYGO-binding) domain of BCL9
ORGANISM(S): Mus musculus (Mouse) 
2020-08-10 | PXD018805 | Pride
Colorectal tumor Transcription Wnt signalling 
UnknownPygopus maintains heart function in aging Drosophila independently of canonical Wnt signaling.
Not available
S-EPMC3871875 | biostudies-literature
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