Sort   by:  
 Page size 
GenomicsNext Generation Sequencing Facilitates Quantitative Analysis of ApoE-/-/SAHHWT, ApoE-/-/SAHH+/- mice and ApoE-/-/SAHH+/- +Betaine mice [WGBS]
Next Generation Sequencing Facilitates Quantitative Analysis of ApoE-/-/SAHHWT, ApoE-/-/SAHH+/- mice and ApoE-/-/SAHH+/- +Betaine mice [WGBS]
PRJNA773078 | ENA
GenomicsNext Generation Sequencing Facilitates Quantitative Analysis of ApoE-/-/SAHHWT, ApoE-/-/SAHH+/- mice and ApoE-/-/SAHH+/- +Betaine mice [RNA-seq]
Next Generation Sequencing Facilitates Quantitative Analysis of ApoE-/-/SAHHWT, ApoE-/-/SAHH+/- mice and ApoE-/-/SAHH+/- +Betaine mice [RNA-seq]
PRJNA773079 | ENA
ProteomicsReversible SAHH inhibitor protects against glomerulonephritis in lupus-prone NZB/NZW F1 mice by modulating α-actinin-4 associated skeleton organization of podocytes
Glomerulonephritis is one of the major complications and causes of death in systemic lupus erythematosus (SLE). Podocytes can be injured in many forms of glomerular disease, characteristic changes are actin cytoskeleton reorganization of involved foot process. ACTN4, encodes α-actinin-4, an actin-bi...
ORGANISM(S): Mus musculus (Mouse) 
2019-11-12 | PXD010059 | Pride
Α-actinin-4 Integrin-linked kinase Podocyte injury Glomerulonephritis Sahh inhibitor Skeleton organization 
TranscriptomicsDissecting the individual contribution of LuxS and autoinducer-2 activity on Streptococcus sanguinis SK36 biofilm formation by chemical, luxS-, and sahH-complementation
In a transcriptome based trail, we figured out that the deletion of luxS has a massive influence to cell growth and metabolism of Streptococcus sanguinis SK36. The biofilm defective luxS deletion mutant was comlemented by transgenic sahH from Pseudomonas aeruginosa. Thus 209 of 216 influenced genes ...
ORGANISM(S): Streptococcus sanguinis SK36 
2012-04-03 | E-GEOD-37007 | biostudies-arrayexpress
Methylation profilingNext Generation Sequencing Facilitates Quantitative Analysis of ApoE-/-/SAHHWT, ApoE-/-/SAHH+/- mice and ApoE-/-/SAHH+/- +Betaine mice [WGBS]
Objective: the next generation sequencing (NGS) has revolutionized the system-based analysis of cellular pathways. The purpose of this study was to explore the mechanism of methylation changes caused by SAHH knockout and dietary betaine addition in atherosclerosis.
ORGANISM(S): Mus musculus 
2021-10-22 | GSE186253 | GEO
TranscriptomicsNext Generation Sequencing Facilitates Quantitative Analysis of ApoE-/-/SAHHWT, ApoE-/-/SAHH+/- mice and ApoE-/-/SAHH+/- +Betaine mice [RNA-seq]
Objective: the next generation sequencing (NGS) has revolutionized the system-based analysis of cellular pathways. The purpose of this study was to explore the mechanism of methylation changes caused by SAHH knockout and dietary betaine addition in atherosclerosis.
ORGANISM(S): Mus musculus 
2021-10-22 | GSE186252 | GEO
UnknownMetformin alters DNA methylation genome-wide via the H19/SAHH axis.
Not available
S-EPMC5415944 | biostudies-literature
UnknownEpigenetic regulation of TXNIP-mediated oxidative stress and NLRP3 inflammasome activation contributes to SAHH inhibition-aggravated diabetic nephropathy.
Not available
S-EPMC8209273 | biostudies-literature
UnknownUltrasound-targeted microbubble technology facilitates SAHH gene delivery to treat diabetic cardiomyopathy by activating AMPK pathway.
Not available
S-EPMC10831940 | biostudies-literature
UnknownCrystallization and preliminary X-ray diffraction analysis of the S-adenosylhomocysteine hydrolase (SAHH) from Thermotoga maritima.
Not available
S-EPMC4231867 | biostudies-literature
Sort   by:  
 Page size