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TranscriptomicsRNAi knock down of export factors in Drosphila Schneider cell lines
Six different endogenous Drosophila proteins (all potential export factors) have been inactivated using RNAi and leptomycin B (LMB) treatments. Five different endogenous Drosophila proteins (all potential export factors) have been targeted by RNAi. The effect on mRNA export is analyzed by monitoring...
ORGANISM(S): Drosophila melanogaster 
2003-05-15 | E-MEXP-4 | biostudies-arrayexpress
UnknownPiggyBac transposon tools for recessive screening identify B-cell lymphoma drivers in mice.
Not available
S-EPMC6440946 | biostudies-literature
GenomicsSimultaneous in vivo enzyme and host engineering through laboratory evolution
Two genetic selection systems that couple metabolite hydroxylation or methylation of small molecules to growth of Escherichia coli are presented in this study. One system targets pterin-dependent hydroxylation (tBPt) while another focuses on S-adenosylmethionine-dependent methylation (SAM). Using ad...
ORGANISM(S): Escherichia coli BW25113 
2019-01-30 | E-MTAB-6779 | biostudies-arrayexpress
UnknownIntroduction and transmission of SARS-CoV-2 lineage B.1.1.7, Alpha variant, in Denmark.
Not available
S-EPMC9064402 | biostudies-literature
UnknownContrasting invertebrate immune defense behaviors caused by a single gene, the Caenorhabditis elegans neuropeptide receptor gene npr-1.
Not available
S-EPMC4827197 | biostudies-literature
UnknownNeuropathologic features associated with basal forebrain atrophy in Alzheimer disease.
Not available
S-EPMC7538215 | biostudies-literature
UnknownIncreased transmissibility of SARS-CoV-2 lineage B.1.1.7 by age and viral load.
Not available
S-EPMC8669007 | biostudies-literature
UnknownWastewater surveillance of SARS-CoV-2 and influenza in preK-12 schools shows school, community, and citywide infections.
Not available
S-EPMC9858235 | biostudies-literature
UnknownGenetic susceptibility for Alzheimer disease neuritic plaque pathology.
Not available
S-EPMC3773291 | biostudies-literature
UnknownSubstrate interaction defects in histidyl-tRNA synthetase linked to dominant axonal peripheral neuropathy.
Not available
S-EPMC5983030 | biostudies-literature
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