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ProteomicsRoles of SNORD115 and SNORD116 ncRNA clusters in neuronal differentiation
Prader-Willi syndrome shows features linked to brain development and hypothalamus-related endocrine abnormalities. The smallest clinical deletions fall within the large (∼650Kb) SNHG14 gene, removing 29 consecutive introns that each generate SNORD116. SNHG14 also includes 48 tandem introns encoding ...
ORGANISM(S): Homo sapiens (Human) 
2025-02-15 | PXD057208 | Pride
Small nucleolar rnas Dia Prader-willi syndrome Snord116 Snord115 
GenomicsTranscriptomic response to adult hypothalamic deletion of Snord116
Transcriptomic response to adult hypothalamic deletion of Snord116
PRJNA415925 | ENA
GenomicsRoles of SNORD115 and SNORD116 ncRNA clusters during neuronal differentiation
Roles of SNORD115 and SNORD116 ncRNA clusters during neuronal differentiation
PRJNA1162504 | ENA
GenomicsRole of Snord116 in pituitary growth hormone deficiency of Prader-Willi Syndrome
Role of Snord116 in pituitary growth hormone deficiency of Prader-Willi Syndrome
PRJNA1472292 | ENA
UnknownPhylogenetic Analysis of the SNORD116 Locus.
Not available
S-EPMC5748676 | biostudies-literature
TranscriptomicsRoles of SNORD115 and SNORD116 ncRNA clusters during neuronal differentiation
In the snoRNA host gene SNHG14, 29 consecutive introns each generate SNORD116, and 48 tandem introns encode SNORD115. Loss of SNORD116 expression, but not of SNORD115, is linked to the neurodevelopmental disease Prader-Willi syndrome. SNORD116 and SNORD115 resemble box C/D small nucleolar RNAs (snoR...
ORGANISM(S): Homo sapiens 
2024-12-05 | GSE277484 | GEO
TranscriptomicsTranscriptomic response to adult hypothalamic deletion of Snord116
Snord116 deletion mouse models recapitulate aspects of the Prader-Willi Syndrome. In this study, we examine the gene expression changes in the mediobasal hypothalamus for mice which have an adult onset deletion of Snord116 in the mediobasal hypothalamus.
ORGANISM(S): Mus musculus 
2017-12-31 | GSE106210 | GEO
UnknownDeletion of the Snord116/SNORD116 Alters Sleep in Mice and Patients with Prader-Willi Syndrome.
Not available
S-EPMC4763347 | biostudies-literature
GenomicsLoss of Snord116 impacts lateral hypothalamus, sleep, and food-related behaviors
Loss of Snord116 impacts lateral hypothalamus, sleep, and food-related behaviors
PRJNA580049 | ENA
TranscriptomicsRole of Snord116 in pituitary growth hormone deficiency of Prader-Willi Syndrome
Prader-Willi Syndrome (PWS) is a complex genetic disorder resulting from the deficiency of several maternally imprinted genes, including SNORD116, in the 15q11-q13 region. Loss of Snord116 in mice recapitulates some of the most salient clinical features of PWS, including growth hormone (GH) deficien...
ORGANISM(S): Mus musculus 
2026-07-30 | GSE333730 | GEO
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