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MetabolomicsThe effect of an NAT10 inhibitor (remodelin) on inflammation-related genes in the treatment of thalamic hemorrhage based on metabolic data

Thalamic hemorrhage is a disease caused by hemorrhage of the thalamic brain parenchyma and is accompanied by an inflammatory response during development. NAT10 has been associated with a wide range of diseases. The aim of this study was to explore the molecular mechanisms associated with the use ...

2025-11-13 | MTBLS11941 | MetaboLights
MetabolomicsDeciphering the Role of N-Acetyltransferase 10 in Thalamic Hemorrhage Through Integrative Multi-Omics and Experimental Validation

Thalamic hemorrhage (TH) is a severe neurological condition, the molecular mechanisms of which are poorly understood, particularly in clinical settings. N-acetyltransferase 10 (NAT10), a regulator of RNA N4-acetylcytidine (ac4C) modification, has been implicated in cell cycle regulation and ident...

2025-11-18 | MTBLS11752 | MetaboLights
ProteomicsThe effect of an NAT10 inhibitor (remodelin) on inflammation-related genes in the treatment of thalamic hemorrhage based on proteomics data
Thalamic hemorrhage is a disease caused by hemorrhage of the thalamic brain parenchyma and is accompanied by an inflammatory response during development. NAT10 has been associated with a wide range of diseases. The aim of this study was to explore the molecular mechanisms associated with the use of ...
ORGANISM(S): Mus musculus (Mouse) 
2025-02-24 | PXD058910 | Pride
Nat10 Thalamic hemorrhage Tmt 
GenomicsA Microglial SPP1–ITGB1 Feedback Loop Sustains Chronic Pain after Thalamic Hemorrhage
A Microglial SPP1–ITGB1 Feedback Loop Sustains Chronic Pain after Thalamic Hemorrhage
PRJNA1512560 | ENA
TranscriptomicsThe effect of an NAT10 inhibitor (remodelin) on inflammation-related genes in the treatment of thalamic hemorrhage based on RNA sequencing data
Thalamic hemorrhage is a disease caused by hemorrhage of the thalamic brain parenchyma and is accompanied by an inflammatory response during development. NAT10 has been associated with a wide range of diseases. The aim of this study was to explore the molecular mechanisms associated with the use of ...
ORGANISM(S): Mus musculus 
2025-11-11 | GSE275066 | GEO
TranscriptomicsA Microglial SPP1–ITGB1 Feedback Loop Sustains Chronic Pain after Thalamic Hemorrhage
This study aimed to characterize transcriptional changes in the thalamic microenvironment following thalamic hemorrhage (TH) and to define how microglial SPP1 contributes to injury-induced cellular responses. Specifically, we compared peri-lesional thalamic tissue from wild-type and Spp1-knockout mi...
ORGANISM(S): Mus musculus 
2026-09-28 | GSE343776 | GEO
GenomicsThe effect of an NAT10 inhibitor (remodelin) on inflammation-related genes in the treatment of thalamic hemorrhage based on RNA sequencing data
The effect of an NAT10 inhibitor (remodelin) on inflammation-related genes in the treatment of thalamic hemorrhage based on RNA sequencing data
PRJNA1149042 | ENA
GenomicsExploring the Mechanism of RIP3 in Thalamic Hemorrhage Based on Animal Models and Transcriptome Sequencing
Exploring the Mechanism of RIP3 in Thalamic Hemorrhage Based on Animal Models and Transcriptome Sequencing
PRJNA1158774 | ENA
UnknownDisrupting interaction of PSD-95 with nNOS attenuates hemorrhage-induced thalamic pain.
Not available
S-EPMC6191031 | biostudies-literature
UnknownFgr contributes to hemorrhage-induced thalamic pain by activating NF-κB/ERK1/2 pathways.
Not available
S-EPMC7605540 | biostudies-literature
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