Viral evasion - OmicsDI

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Lab affiliation

Cambridge Institute for Medical Research, University of Cambridge, Hills Road, Cambridge, CB2 0XY, UK (7)
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Cambridge Institute for Medical Research, University of Cambridge, Hills Road, Cambridge, CB2 0XY (1)
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Clinical Proteomics Medizinisches Proteom-Center Ruhr-University Bochum (1)
Core Unit Proteomics, Berlin Institute of Health at Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, 13125 Berlin, Germany (1)
Dalian medical university (1)
Department of Cellular and Molecular Pharmacology, UCSF (1)
Department of Genetics, Immunogenomics and Molecular Pathogenesis Group, UIC Zoonoses and Emergent Diseases ENZOEM, University of Córdoba, Córdoba, Spain (1)
Department of Preventive Medicine, Public Health and Microbiology, Universidad Autónoma de Madrid, Madrid, Spain Department of Microbiology, Icahn School of Medicine atMount Sinai, New York, New York, USA. Global Health and Emerging Pathogens Institute, Icahn School of Medicine at Mount Sinai, New York, New York, USA (1)
Dept. Medical Microbiology, Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, Nijmegen, the Netherlands (1)
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Harald zur Hausen Institute of Virology, Erlangen, Germany (1)
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Institute of Organic Chemistry and Biochemistry of the Czech Academy of Sciences, Flemingovo namesti 542/2, 16000, Prague, Czech Republic (1)
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Interdisciplinary research center HALOmem, Institute of Biochemistry and Biotechnology, Charles Tanford Protein Center, Martin Luther University Halle-Wittenberg, Halle, Germany (1)
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Quantitative Biosciences Institute (QBI), University of California, San Francisco, San Francisco, CA USA J. David Gladstone Institutes, San Francisco, San Francisco, CA, USA Department of Bionengineer and Therapeutic Sciences, University of California, San Francisco, San Francisco, CA, USA (1)

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Transcription profiling of Jurkat CF4+ T cells transfected with simian immunodeficiency virus Nef cells leads to a molecular profile of viral persistence and immune evasion.

Jurkat T cell line was transfected with SIV Nef controlled by an inducible promoter system. Nef was induced by addition of 10 micromolar pronasterone A and gene expression values were determined after 24 hrs. Control Jurkat cells were untransfected and treated with 10 micromolar pronasterone A and g...

ORGANISM(S): Homo sapiens

2007-10-26 | E-GEOD-4785 | biostudies-arrayexpress

Global changes in Staphylococcus aureus gene expression in human blood provide insight into mechanisms of immune evasion and virulence

Staphylococcus aureus is a leading cause of bloodstream infections worldwide. In the United States, many of these infections are caused by a strain known as USA300. Although progress has been made, our understanding of the S. aureus molecules that promote bacteremia and survival in human blood is ...

ORGANISM(S): Staphylococcus aureus

2011-08-25 | E-GEOD-25454 | biostudies-arrayexpress

Structure and mechanism of a novel cytomegaloviral DCAF mediating interferon antagonism

Human cytomegalovirus (CMV) is a highly relevant pathogen, and its rodent counterparts serve as common infection models. Global proteome profiling of rat CMV (RCMV)-infected cells uncovered a pronounced loss of the transcription factor STAT2, which is crucial for interferon signalling. Deletion muta...

ORGANISM(S): Cellular Organisms

2022-12-15 | PXD033366 | JPOST Repository

Selective modulation of cell surface proteins during vaccinia infection: a resource for identifying viral immune evasion strategies

The interaction between immune cells and virus-infected targets involves multiple plasma membrane (PM) proteins. A systematic study of PM protein modulation by vaccinia virus (VACV), the paradigm of host regulation, has the potential to reveal not only novel viral immune evasion mechanisms, but also...

ORGANISM(S): Homo sapiens (Human) Vaccinia virus WR Vaccinia virus

2022-06-01 | PXD033407 | Pride

Chronic viral mimicry induction following p53 loss promotes immune evasion [scRNASeq]

PRJNA896215 | ENA

Chronic viral mimicry induction following p53 loss promotes immune evasion [CUTandRUN]

PRJNA894548 | ENA

Transcription profiling of mouse cell lines immune sensitive P0 and immune resistant P3 identifies a tumor immune evasion mechanism using P0 and P3 cell lines

The emergence of immune resistance variants during immunotherapy is poorly understood. We generated a highly immune resistant cell line (P3) from a susceptible cell line (P0) by subjecting it to 3 rounds of in vivo immune selection. Subsequently, microarray analysis of P0 and P3 was performed to i...

ORGANISM(S): Mus musculus

2008-06-12 | E-GEOD-2774 | biostudies-arrayexpress

A gut-brain-body circuit mediates immune evasion in viral infection

PRJNA1212042 | ENA

Chronic viral mimicry induction following p53 loss promotes immune evasion [Mouse RNASeq]

PRJNA896208 | ENA

Chronic viral mimicry induction following p53 loss promotes immune evasion [STIC RNASeq]

PRJNA896225 | ENA

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