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TranscriptomicsGene expression of human isogenic trisomy 21 induced pluripotent stem cells and derived neurons
Down syndrome (trisomy 21) is the most common genetic cause of intellectual disability, but the precise molecular mechanisms underlying impaired cognition remain unclear. Elucidation of these mechanisms has been hindered by the lack of a model system that contains full trisomy of chromosome 21 (Ts21...
ORGANISM(S): Homo sapiens 
2013-07-09 | E-GEOD-48611 | biostudies-arrayexpress
UnknownMiR-425-5p intervenes in autoimmune myocarditis by regulating Treg cell differentiation through NRAS.
Not available
S-EPMC12106460 | biostudies-literature
ProteomicsMass spectrum data for the manuscript “oocyte-derived microvilli control female fertility by optimizing the ovarian follicle selection in mice”
To clarify our hypothesis and investigate the functional role of Oo-Mv in folliculogenesis, we isolated the ZP containing Oo-Mv from oocytes and performed mass spectrometry (MS) analysis to identify potential regulators of the formation of Oo-Mv.
ORGANISM(S): Mus Musculus 
2021-03-23 | PXD024931 |
ProteomicsQuantitative acetylomics reveals dynamics of protein lysine acetylation in mouse livers during aging and upon the treatment of nicotinamide mononucleotide
Acetylome of livers of aging mouse and NMN treatment mouse
ORGANISM(S): Mus Musculus 
2022-02-23 | PXD031851 |
ProteomicsRapamycin targets RBBP7 to regulate histone H4 acetylation and suppress hepatocellular carcinoma.
Identifying therapeutic protein targets is challenging due to unpredictable druggability. Exploring multi-purpose drug targetomes, such as that of rapamycin, offers a valuable strategy for target discovery. Here, using chemical proteomics, we identified the histone-binding protein RBBP7 as a previou...
ORGANISM(S): Homo Sapiens 
2026-07-13 | PXD080960 |
ProteomicsAML cell line THP-1 shControl VS THP-1 shPSMB10
In the THP-1 cell line, knockdown of PSMB10 expression was performed using lentivirus, with a control group set up.
ORGANISM(S): Homo Sapiens 
2024-12-10 | PXD058714 |
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