The FGF4–integrin β1 axis restores directed collective migration to accelerate diabetic wound regeneration [ChIP-Seq].

ABSTRACT: This dataset was generated to investigate the mechanism by which Fibroblast Growth Factor 4 (FGF4) regulates the binding of C-FOS to the ITGB1 promoter. ChIP sequencing was performed on high glucose-induced HacaT cells, which were divided into low glucose, high glucose, and high glucose plus FGF4 treatment groups.

ORGANISM(S): Homo sapiens

PROVIDER: GSE318682 | GEO | 2026/05/01

REPOSITORIES: GEO