S-nitrosylation in Shank3 mutated mice

ABSTRACT: We tested the hypothesis that a mutated gene associated with human ASD would generate downstream effects in critical proteins that lead to modification of synaptic functions.

INSTRUMENT(S):

ORGANISM(S): Mus Musculus (mouse)

TISSUE(S): Brain

SUBMITTER: Haitham Amal  

LAB HEAD: Prof. Steven R. Tannenbaum

PROVIDER: PXD006907 | Pride | 2018-10-02

REPOSITORIES: Pride