twist1 - OmicsDI

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Assistant Professor of Biological Sciences Augusta University C2014 Science Hall 2500 Walton Way, Augusta-GA (1)
Department of Pediatrics, Department of Cell Biology, Neurobiology and Anatomy Medical College of Wisconsin, Milwaukee, WI, United States (1)
Pediatric Hematology-Oncology Research Laboratory, Woman-Mother-Child Department, Lausanne University Hospital and University of Lausanne, Switzerland. (1)
Prof. Dr. Stefan Mundlos Max-Planck-Institut für molekulare Genetik Ihnestraße 63-73 14195 Berlin Germany (1)
Research Institute for Complex Issues of Cardiovascular Diseases (1)
School of Pharmacy, Lanzhou University, Lanzhou 730000, China (1)
The Shraga Segal Department of Microbiology, Immunology and Genetics,National Institute for Biotechnology in the Negev, Ben-Gurion University of the Negev, P.O.B. 653, Be'er-Sheva 84105, Israel (1)

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6x(2)H labeled dimethylated L-arginine (1)

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Functional analysis of TWIST1 domains regulating smooth muscle cell phenotype

TWIST1, a bHLH transcription factor, regulates mesenchymal specification, differentiation, proliferation and migration during development and in diseases such as cancer. More recently, genome-wide association studies have identified TWIST1 as a causal gene that increases risk for multiple vascular ...

ORGANISM(S): Homo sapiens

2025-10-14 | E-MTAB-15722 | biostudies-arrayexpress

The expression level of microRNA after knockdown of Twist1 or Bmi1

To screen the microRNA regulated by Twist1 and Bmi1 Establish stable transfectants of pSUPER-sh-Twist1 or pSUPER-sh-Bmi1 in OECM1 cells and analyze the miRNA expression level of by microRNA microarray. OECM1 transfected with pSUPER-sh-scr was used as a control experiment.

ORGANISM(S): Homo sapiens

2013-02-11 | E-GEOD-29586 | biostudies-arrayexpress

MET after transient Twist activation results in de novo gain of malignant traits

During Epithelial-Mesenchymal Transition (EMT), apical-basal polarized epithelial cells are converted to front-to-back polarized mesenchymal cells that only form loose cell-cell adhesions. These phenotypic changes are accompanied by acquisition of increased motility and invasiveness. EMT programs ar...

ORGANISM(S): Homo sapiens

2015-07-02 | E-GEOD-61206 | biostudies-arrayexpress

Architecture of Epigenetic Reprogramming Following Twist1 Mediated Epithelial-Mesenchymal Transition [RNA-seq]

Purpose: to characterize epigenetic changes following Twist1 mediated Epithelial-Mesenchymal Transition in human Methods: we characterized the epigenetic and transcriptome landscapes using whole genome transcriptome analysis by RNA-seq, DNA methylation by digital restriction enzyme analysis of meth...

ORGANISM(S): Homo sapiens

2013-12-10 | E-GEOD-53024 | biostudies-arrayexpress

TWIST1-induced microRNA-424 drives an intermediate epithelial-to-mesenchymal transition that opposes metastasis

Using a TWIST1-inducible epithelial-to-mesenchymal transition (EMT) model in HMLE cells, miRNA changes were profiled at different time points during an active EMT. This experiment includes a total of 12 samples consisting of 2 experimental groups at 2 matching time points (day 4, day 12), each with ...

ORGANISM(S): Homo sapiens

2015-02-26 | E-GEOD-58560 | biostudies-arrayexpress

Architecture of Epigenetic Reprogramming Following Twist1 Mediated Epithelial-Mesenchymal Transition [ChIP-seq]

ORGANISM(S): Homo sapiens

2013-12-10 | E-GEOD-53023 | biostudies-arrayexpress

Architecture of Epigenetic Reprogramming Following Twist1 Mediated Epithelial-Mesenchymal Transition [DREAM-seq]

ORGANISM(S): Homo sapiens

2013-12-10 | E-GEOD-53025 | biostudies-arrayexpress

Effect of Twist-box mutation on gene expression induced by Twist1 overexpression

Twist1 is a transcription factor that induces EMT and drives metastasis in prostate cancer. We examined global gene expression in Myc-CaP mouse prostate cancer cells following overexpression of Twist1 and the Twist1 mutants F191G, AQA, and DQD. 15 total samples were analysed, with 3 replicates of 5 ...

ORGANISM(S): Mus musculus

2013-08-20 | E-GEOD-50002 | biostudies-arrayexpress

Identification of TWIST1 transcriptional targets in the cranial mesoderm [ChIP-Seq]

TWIST1, a basic helix-loop-helix transcription factor is essential for the development of cranial mesoderm and cranial neural crest-derived craniofacial structures. Our previous work showed that, in the absence of TWIST1, some cells within the cranial mesoderm adopt an abnormal epithelial configurat...

ORGANISM(S): Canis lupus familiaris

2016-08-23 | E-GEOD-80662 | biostudies-arrayexpress

TWIST1 methylation by SETD6 selectively antagonizes LINC-PINT expression in Glioblastoma multiforme

Glioblastoma multiforme (GBM) is the most common and aggressive malignant brain tumor among adults, which is characterized by high invasion, migration and proliferation abilities. One important process that contributes to the invasiveness of GBM is the epithelial to mesenchymal transition (EMT). EMT...

ORGANISM(S): Homo sapiens (Human)

2022-08-12 | PXD029361 | Pride

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