Alpha-galactosidase a

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CHEBI ID

CHEBI:57790 (1)
CHEBI:80153 (1)
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CHEBI:79612 (1)
CHEBI:79444 (1)
CHEBI:81173 (1)
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CHEBI:72449 (1)
CHEBI:49124 (1)
CHEBI:34789 (1)
CHEBI:32239 (1)
CHEBI:73113 (1)
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CHEBI:24424 (1)
CHEBI:34462 (1)
CHEBI:80824 (1)
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CHEBI:473992 (1)
CHEBI:67357 (1)
CHEBI:38504 (1)
CHEBI:8865 (1)
CHEBI:23267 (1)
CHEBI:50720 (1)
CHEBI:35024 (1)
CHEBI:71419 (1)

Metabolite Name

Morphine-3-glucuronide (2)
11-Ketoetiocholanolone (2)
17¦Á-Ethynylestradiol (1)
¦Á-Cyclodextrin (1)
8-iso-15-keto Prostaglandin F2¦Á (1)
3-(2,4-difluoroanilino)-2-(2-thienylsulfonyl)acrylonitrile (1)
2-(3-chlorophenyl)-2-hydroxyacetic acid (1)
6-methyl-5-nitroquinoline (1)
(+/-)8(9)-DiHETE (1)
(6R,S)-5, 10-Methylene-5,6,7,8-tetrahydrofolic acid (1)
Mephedrone-d3 (1)
4-(2,3-dihydrobenzo[b]furan-5-yl)pyrimidine-2-thiol (1)
Lapatinib Ditosylate (1)
(+/-)5-iPF2¦Á-VI (1)
2-[2-(5-bromo-2-pyridyl)diaz-1-enyl]-5-(diethylamino)phenol (1)
N1-(4-morpholinophenyl)-2-methylbut-2-enamide (1)
2-cyano-3-(4-hydroxyphenyl)acrylamide (1)
3-chloro-2,6-dimethoxybenzoic acid (1)
2-Keto-4-methylthiobutyric acid (1)
19(R)-Hydroxy prostaglandin F2¦Á (1)
15(R),19(R)-Hydroxy prostaglandin F1¦Á (1)
10-Formylpteroic acid (1)
9-[(2-hydroxyethoxy)methyl]-1,9-dihydro-6H-purin-6-one (1)
3-oxoindane-1-carboxylic acid (1)
5-trans-Prostaglandin E2 (1)
DY131 (1)
Prostaglandin F1¦Â (1)
DL-¦Á-Methoxyphenylacetic acid (1)
Prostaglandin F3¦Á (1)
2,3-Dinor-8-epi-prostaglandin F2¦Á (1)

First Public Date

2014 (5)
2022 (4)
2026 (3)
2025 (3)
2023 (3)
2024 (2)
2019 (2)
2017 (2)
2015 (2)
2018 (1)
2016 (1)

Classification

bacteria (2)
fungi (1)

Study type

Transcription profiling by array (32)
RNA-seq of coding RNA (6)
Unknown experiment type (2)
ChIP-seq (2)
RNA-seq of coding RNA from single cells (2)
ChIP-chip by tiling array (1)
ChIP-chip by array (1)
Genotyping by array (1)

Release Date

2021 (100)
2023 (83)
2024 (77)
2022 (75)
2020 (66)
2025 (54)
2019 (54)
2018 (54)
2017 (43)
2015 (39)
2016 (38)
2014 (37)
2012 (29)
2013 (20)
2010 (20)
2011 (15)
2009 (14)
2008 (11)
2007 (11)
2026 (9)
2002 (8)
2001 (8)
2006 (6)
2005 (6)
1999 (6)
2003 (5)
2004 (4)
1998 (3)
2000 (1)

Lab affiliation

National Heart and Lung Institute, Imperial College London, London, UK (3)
Analytics Core Facility Essen (ACE), Chemische Biologie, Universität Duisburg-Essen, ZMB, Germany (1)
Copenhagen Center for Glycomics (CCG), Department of Cellular and Molecular Medicine, University of Copenhagen (1)
Copenhagen University (1)
Department of Biochemistry Center for Biomedical Mass Spectrometry Boston University School of Medicine (1)
Department of Cellular and Molecular Medicine, University of Copenhagen (1)
Department of Chemistry and Biomolecular Sciences Macquarie University Sydney, NSW 2109 Australia (1)
Department of Medicine, Haukeland University Hospital, Bergen, Norway (1)
Department of Pediatrics, Department of Cell Biology, Neurobiology and Anatomy Medical College of Wisconsin, Milwaukee, WI, United States (1)
Department of Plant Bioscience, Life and industry Convergence Research Institute, Pusan National University, Miryang, 50463, Republic of Korea (1)
Federal University of Rio de Janeiro (1)
III. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany (1)
Instituto de Investigaciones Sanitarias de Santiago de Compostela (1)
Jiang Pingping Laboratory, School of Basic Medical Sciences, Zhejiang University School of Medicine (1)
National Heart and Lung Institute, Imperial College London, United Kingdom (1)
Research Unit for Rare Diseases, Department of Paediatrics and Inherited Metabolic Disorders, First Faculty of Medicine, Charles University in Prague and General University Hospital in Prague, Czech Republic (1)
University Hospital Cologne (1)

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Proteomics analysis of podocytes with alpha-galactosidase knockdown

We subjected human podocytes with and without alpha-Galactosidase knockdown to proteomic analysis.

ORGANISM(S): Homo sapiens (Human)

2018-10-17 | PXD007080 | Pride

New α-galactosidase-inhibiting aminohydroxycyclopentanes.

Not available

S-EPMC9029992 | biostudies-literature

Proteome analysis of HEK293 cells stably expressing wild-type and mutated AGAL enzyme

Fabry disease is an X-linked condition caused by variants of the GLA gene that result in the absence or enzymatic deficiency of alpha-galactosidase A (AGAL). This enzyme defect leads to lysosomal storage of globotriaosylceramide (Gb3Cer) in a variety of cell types throughout the body and manifests a...

ORGANISM(S): Homo sapiens (Human)

2025-11-06 | PXD033936 | Pride

Alpha-galactosidase a Fabry disease

Bioconversion of Beet Molasses to Alpha-Galactosidase and Ethanol.

Not available

S-EPMC6416216 | biostudies-literature

Valuation of agro-industrial wastes as substrates for heterologous production of α-galactosidase.

Not available

S-EPMC6122717 | biostudies-literature

The New Pharmacological Chaperones PBXs Increase α-Galactosidase A Activity in Fabry Disease Cellular Models.

Not available

S-EPMC8699827 | biostudies-literature