Angiotensinogen

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- (1)
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Metabolite Name

Tulathromycin A (2)
N-Acetyl-L-glutamine (2)
Cer(t18:0/16:0) (2)
Cortolone (2)
16-Hydroxyhexadecanoic acid (2)
Hexadecanedioic acid (2)
Uric acid (2)
Cytidine (2)
5-methoxy-2-(2-methoxyphenyl)chromen-4-one (1)
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2-hydroxynaphthalene-1-carbaldehyde (1)
5,7-dihydroxy-2-(4-hydroxyphenyl)-8-(3-methylbut-2-enyl)chromen-4-one (1)
5,8-dihydroxy-2-phenyl-2,3-dihydrochromen-4-one (1)
2-[({3-[3,4-dihydroxy-5-(3,4,5-trihydroxybenzoyloxy)benzoyloxy]-4,5-dihydroxyphenyl}(hydroxy)methylidene)amino]acetic acid (1)
2-amino-4'-hydroxy-Propiophenone (1)
Alpha-Fluoro-beta-ureidopropionic acid (1)
(4S,5S,10R)-4-Hydroxy-5-methoxy-10-methyl-2,7-oxecanedione (1)
3,4,5-trihydroxy-6-[(2-oxo-2H-chromen-4-yl)oxy]oxane-2-carboxylic acid (1)
5-(hydroxymethyl)- 2-Furancarboxylic acid (1)
Methyl (E)-11-(5-carbamoyl-4-oxo-1H-pyridin-2-yl)undec-10-enoate (1)
(2R,3S,4S,5R,6R)-2-methylol-6-phenethoxy-tetrahydropyran-3,4,5-triol (1)
8-{(1S,5R)-4-Oxo-5-[(2Z)-2-penten-1-yl]-2-cyclopenten-1-yl}octanoic acid (1)
Depo-Medrol (1)
3-(1,3-benzodioxol-5-yl)propane-1,2-diol (1)
3-hydroxy-N-(2-oxooxolan-3-yl)octanamide (1)
3,7-dihydroxy-2-(4-hydroxyphenyl)chromen-4-one (1)
2-Aminoheptane (1)
2-(1-{[(1-{3-[(E)-2-(7-chloroquinolin-2-yl)ethenyl]phenyl}-3-hydroxy-3-[2-(2-hydroxypropan-2-yl)phenyl]propyl)sulfanyl]methyl}cyclopropyl)acetic acid (1)
3-[2-[2-(carboxymethoxy)ethoxy]ethoxymethyl]heptanoic acid (1)
8-(2,3-dihydroxy-3-methylbutyl)-5,7-dimethoxy-2-phenyl-2,3-dihydrochromen-4-one (1)

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2014 (6)
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Study type

Transcription profiling by array (10)
Animal - One-color microarray (1)
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Release Date

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1996 (1)

Lab affiliation

Center for Environmental and Human Toxicology Department of Physiological Sciences & College of Medicine College of Veterinary Medicine University of Florida (1)
Department of Molecular and Developmental Medicine, University of Siena, Italy (1)
Department of Pediatrics University of Maryland, School of Medicine (1)
Department of Pediatrics, Clinical Sciences Lund, Lund University, 221 85, Lund, Sweden (1)
Target Discovery Institute Nuffield Department of Medicine University of Oxford Roosevelt Drive Oxford OX3 FZ UK Tel: +44 (0) 1865 612 921 https://www.tdi.ox.ac.uk/research/research/tdi-mass-spectrometry-laboratory (1)
University of Maryland School of Medicine, Center for Vascular and Inflammatory Diseases (1)

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Angiotensinogen and C3 compete for renin-induced complement activation

Demonstration of renin cleavage of C3. Angiotensinogen and C3 compete for renin-induced complement activation.

ORGANISM(S): Homo sapiens (Human)

2025-04-18 | PXD053116 | Pride

C3 Renin Angiotensinogen Lc-ms/ms

Genome-wide analysis of gene expression in white adipose tissue in angiotensinogen overexpression transgenic and wild type male B6 mice

PRJNA627708 | ENA

Genome-wide analysis of gene expression in white adipose tissue in angiotensinogen overexpression transgenic and wild type male B6 mice

Investigation of differential gene regulation with overexpression of angiotensinogen in white adipose tissue. We hypothesized that angiotensinogen overexpression will disrupt metabolic homeostasis of adipocytes leading to obesity and related chronic diseases. Results indicate that angiotensinogen ov...

ORGANISM(S): Mus musculus

2020-07-15 | GSE149231 | GEO

A redox switch in angiotensinogen modulates angiotensin release.

Not available

S-EPMC3024006 | biostudies-literature

Allele-specific expression of angiotensinogen in human subcutaneous adipose tissue.

Not available

S-EPMC3718031 | biostudies-literature

Hepatocyte angiotensinogen deletion protects against Western diet-induced adiposity and hepatic steatosis under thermoneutral housing

PRJNA1494419 | ENA

Angiotensinogen and C3 compete for renin-induced complement activation.

Not available

S-EPMC11999838 | biostudies-literature

Determination of plasma and urinary angiotensinogen levels in rodents by newly developed ELISA.

Not available

S-EPMC2610404 | biostudies-literature

AT1 receptor mediated augmentation of intrarenal angiotensinogen in angiotensin II-dependent hypertension.

Not available

S-EPMC2637079 | biostudies-literature

Involvement of the brain renin-angiotensin system in the effects of maternal separation

The postnatal development of the mouse is characterized by a stress hyporesponsive period (SHRP), where basal corticosterone levels are low and responsiveness to mild stressors is reduced. Maternal separation is able to disrupt the SHRP and is widely used to model early trauma. In this study we aime...

ORGANISM(S): Mus musculus

2010-05-16 | E-GEOD-14687 | biostudies-arrayexpress

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