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MetabolomicsPrimary Osteocyte Supernatants Metabolomic Profiling of Two Transgenic Mice with Connexin43 Dominant Negative Mutants

Osteocytes could release some small molecules (≤ 1 kDa) through gap junctions and hemichannels to extracellular environment, such as prostaglandin E2 (PGE2), nitric oxide (NO) and adenosine triphosphate (ATP), which play key roles in transferring signals between bone cells and other tissue cells....

2021-04-14 | MTBLS2358 | MetaboLights
ProteomicsPolydisperse molecular architecture of connexin 26/30 heteromeric hemichannels revealed by AFM imaging
Gap junctions are arrays of intercellular channels formed by two hemichannels docked end-to-end. Each hemichannel is a hexamer containing the same or different connexin (Cx) isoform. While homomeric Cxs forms have been largely described functionally and structurally, lesser is known about heteromeri...
ORGANISM(S): Homo sapiens (Human) 
2020-09-04 | PXD020633 | Pride
Lc-ms/ms Connexin 
GenomicsDysregulated genes in connexin 30 deficient mice microglia [EAE]
Dysregulated genes in connexin 30 deficient mice microglia [EAE]
PRJNA448529 | ENA
GenomicsDysregulated genes in connexin 30 deficient mice microglia [naive]
Dysregulated genes in connexin 30 deficient mice microglia [naive]
PRJNA448530 | ENA
GenomicsDysregulated genes in connexin 30 deficient mice microglia
Dysregulated genes in connexin 30 deficient mice microglia
PRJNA448527 | ENA
TranscriptomicsTranscription profiling of mouse with ablation of connexin 30 in the stria vascularis
Effect of the ablation of connexin 30 in the stria vascularis
ORGANISM(S): Mus musculus 
2007-04-16 | E-MEXP-558 | biostudies-arrayexpress
UnknownConnexin 30 controls astroglial polarization during postnatal brain development.
Not available
S-EPMC5869003 | biostudies-literature
GenomicsConnexin 30 deficiency ameliorates the disease progression of amyotrophic lateral sclerosis model mice by suppressing glial inflammation.
Connexin 30 deficiency ameliorates the disease progression of amyotrophic lateral sclerosis model mice by suppressing glial inflammation.
PRJNA882765 | ENA
ProteomicsCo-IP (AP-MS) of Connexin-43 - Gap junction protein Connexin-43 is a direct transcriptional regulator of N-cadherin in vivo
Co-IP Experiment Bait: Cx43-Flag Cell line: Hela Related Publication: Gap Junction protein Connexin-43 is a direct transcriptional regulator of N-cadherin in vivo, Maria Kotini, Elias Barriga, Jonathan Leslie, Marc Gentzel, Verena Rauschenberger, Alexandra Schambony, and Roberto Mayor
ORGANISM(S): Homo sapiens (Human) 
2018-10-16 | PXD010870 | Pride
Ap-ms Hela Co-ip 
TranscriptomicsDysregulated genes in connexin 30 deficient mice microglia [EAE]
Objective: Connexins are known to compose gap junctions by a pair of hemichannels connected in a head to head configuration. Each hemichannel is composed by hexameric cluster of connexins. There are 20 members of connexins known as connexin family in mice, and are expressed on each cell surface in d...
ORGANISM(S): Mus musculus 
2018-04-04 | GSE112620 | GEO
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