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TranscriptomicsTranscription profiling of gastrocneumius muscle from Emerin and Lamin A/C knockout mice after recovery from cardiotoxin injection to study muscle regeneration
Study muscle regeneration in Emerin and Lamin AC KO mice. Gastrocnemius muscles were injected with cardiotoxin. Muscles were isolated at different time points. Keywords: Time-Series The specific aim is to generate staged muscle degeneration/regeneration of Emerin deficient and Lamin A/C deficient mi...
ORGANISM(S): Mus musculus 
2006-07-12 | E-GEOD-5304 | biostudies-arrayexpress
ProteomicsIdentification of PP2A-B55 targets in Drosophila uncovers regulation of Emerin during nuclear envelope reassembly
Entry into mitosis is triggered by mitotic kinases that phosphorylate multiple proteins to induce profound cellular changes including nuclear envelope breakdown, chromosome condensation and spindle assembly. Conversely, mitotic exit requires protein dephosphorylation as cells return to their interph...
ORGANISM(S): Drosophila melanogaster (Fruit fly) 
2023-08-10 | PXD041032 | Pride
Otefin Pp2a Baf Lamin Nuclear envelope Mitotic exit Mitosis Emerin 
ProteomicsIdentification of PP2A-B55 targets in Drosophila uncovers regulation of Emerin during nuclear envelope reassembly
Entry into mitosis is triggered by mitotic kinases that phosphorylate multiple proteins to induce profound cellular changes including nuclear envelope breakdown, chromosome condensation and spindle assembly. Conversely, mitotic exit requires protein dephosphorylation as cells return to their interph...
ORGANISM(S): Drosophila melanogaster (Fruit fly) 
2023-08-10 | PXD041030 | Pride
Otefin Pp2a Baf Lamin Nuclear envelope Mitotic exit Mitosis Emerin 
ProteomicsIdentification of PP2A-B55 targets in Drosophila uncovers regulation of Emerin during nuclear envelope reassembly
Entry into mitosis is triggered by mitotic kinases that phosphorylate multiple proteins to induce profound cellular changes including nuclear envelope breakdown, chromosome condensation and spindle assembly. Conversely, mitotic exit requires protein dephosphorylation as cells return to their interph...
ORGANISM(S): Drosophila melanogaster (Fruit fly) 
2023-08-10 | PXD041091 | Pride
Otefin Pp2a Baf Lamin Nuclear envelope Mitotic exit Mitosis Emerin 
UnknownThe molecular basis of emerin-emerin and emerin-BAF interactions.
Not available
S-EPMC4163644 | biostudies-literature
GenomicsTranscriptional contribution of nuclear lamina protein emerin and BAF in Drosophila ovaries
Transcriptional contribution of nuclear lamina protein emerin and BAF in Drosophila ovaries
PRJNA705972 | ENA
GenomicsEDMD-causing emerin mutant myogenic progenitors exhibit impaired differentiation using similar mechanisms
EDMD-causing emerin mutant myogenic progenitors exhibit impaired differentiation using similar mechanisms
PRJNA638664 | ENA
TranscriptomicsEDMD-causing emerin mutant myogenic progenitors exhibit impaired differentiation using similar mechanisms
RNA sequencing was performed on proliferating and differentiating emerin-null myogenic progenitors expressing emerin and EDMD-causing emerin mutants to identify molecular pathways implicated in Emery-Dreifuss Muscular Dystrophy.
ORGANISM(S): Mus musculus 
2020-07-01 | GSE152226 | GEO
ProteomicsShared and distinctive neighborhoods of emerin and LBR revealed by proximity labeling and quantitative proteomics
Emerin and LBR are abundant transmembrane proteins of the nuclear envelope (NE) that are concentrated at the inner nuclear membrane (INM). Although both proteins interact with chromatin and nuclear lamins, they have distinctive biochemical and functional properties. Here we have deployed proximity...
ORGANISM(S): Mus Musculus (ncbitaxon:10090) 
2022-05-02 | MSV000089356 | MassIVE
GenomicsEmerin loss of function inhibits MyoD-driven differentiation of human iPSCs into skeletal myotubes
Emerin loss of function inhibits MyoD-driven differentiation of human iPSCs into skeletal myotubes
PRJNA1514080 | ENA
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