High-dose radiation

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unidentified (1)
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Repository

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Disease

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Instrument Platform

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UNIPROT ID/AC

P53152 (1)
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CHEBI ID

CHEBI:422 (3)
CHEBI:16977 (3)
CHEBI:15428 (3)
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CHEBI:17295 (3)
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CHEBI:15859 (2)

Metabolite Name

Maleimide (2)
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First Public Date

2025 (37)
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2018 (13)
2020 (10)
2021 (9)
2019 (9)
2015 (8)
2016 (5)
2017 (4)

Modelling Approach

Ordinary differential equation model (2)

Study status

Completed (8)
Not provided (4)
Withdrawn (2)
Terminated (2)
Active, not recruiting (1)
Not yet recruiting (1)

Lab affiliation

EDyP (2)
Beijing Key Laboratory for Radiobiology, Beijing Institute of Radiation Medicine, Beijing 100850, P.R. China (1)
Columbia University Irving Medical Center, New York, NY, USA (1)
DNA damage responses and cancer lab Department of Human Genetics Leiden University Medical Center Eithovenweg 20 2333ZC Leiden The Netherlands (1)
DNA damage responses and cancer lab Department of Human Genetics Leiden University Medical Center Eithovenweg 20 2333ZC Leiden The Netherlands (1)
Department of Experimental Radiation Oncology, The University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA (1)
Department of Neurobiology, Harvard Medical School; Boston, USA. (1)
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Helmholtz Zentrum München - German Research Center for Environmental Health, Institute of Radiation Biology, Neuherberg, Germany (1)
Institute for Radiation Biology, Helmholtz Center Munich, German Research Center for Environmental Health GmbH, Neuherberg, Germany (1)
Institute of Clinical Medicine, University of Tartu, Tartu, Estonia (1)
Institute of Radiation Biology, Radiation Proteomics, Helmholtz Zentrum München, German Research Center for Environmental Health GmbH (HMGU), 85764 Neuherberg, Germany (1)
PSE-SANTE/SERAMED, LRMED, IRSN (1)
Radiation Signaling Group, Radiation Biology & Health Sciences Division, Bio-Science Group, Bhabha Atomic Research Centre (BARC), Mumbai - 400 085, India and Homi Bhabha National Institute (HBNI), Mumbai - 400 094, India (1)
Shandong University, Shandong Provincial Hospital, Jinan, Shandong, 250021, P.R. China (1)
State Key Laboratory of Proteomics, National Center for Protein Sciences Beijing, Beijing Proteome Research Center (1)
Surgery, University of Florida (1)
UMR CNRS 8200, Institut Gustave Roussy, Université Paris-Saclay, Villejuif, France (1)
University Medicine Greifswald Interfaculty Institute for Genetics and Functional Genomics Department of Functional Genomics Friedrich-Ludwig-Jahn-Str. 15a 17475 Greifswald, Germany (1)

Keyword

Stage III rectal cancer (6)
Adenocarcinoma of the rectum (6)
Stage II rectal cancer (4)
Capecitabine (4)
Radiation (2)
Stage IV gastric cancer (2)
Recurrent gastric cancer (2)
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Enoxaparin (1)
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Dosimetry (1)
Ursodeoxycholic Acid (1)
Irradiation (1)
Bortezomib (1)
Tremelimumab (1)
Poly ICLC (1)
Adult primary hepatocellular carcinoma (1)
Liver metastases of colorectal cancer (1)
Metastases of melanoma or renal cancer (1)
Pentoxifylline (1)
Adenocarcinoma of the extrahepatic bile duct (1)

IsPrivate

False (2)

Modification

2x(15)N labeled L-lysine (2)
4 (2)
4x(15)N labeled L-arginine (2)

Study type

Transcription profiling by array (227)
RNA-seq of coding RNA (26)
Interventional (18)
Comparative genomic hybridization by array (5)
Methylation profiling by array (5)
Animal - One-color microarray (4)
ChIP-seq (4)
Methylation profiling by high throughput sequencing (4)
Other (3)
MicroRNA profiling by array (3)
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Genotyping by array (1)
MicroRNA profiling by high throughput sequencing (1)
Proteomic profiling by array (1)
RNA-seq of coding RNA from single cells (1)
RNA-seq of total RNA (1)

Release Date

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2015 (172)
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2013 (117)
2014 (115)
2012 (88)
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2011 (69)
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2008 (23)
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1999 (1)

Location

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China (1)
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Long-term low-dose ionizing radiation induced chromosome-aberration-specific metabolic phenotype changes in radiation workers

Long-term low-dose ionizing radiation (LLIR) widely exists in human life and has been confirmed to have potential pathogenic effects on cancer and cardiovascular diseases. However, it is technically and ethically unfeasible to explore LLIR-induced phenotypic changes in the human cohort, leading t...

2022-04-07 | MTBLS3854 | MetaboLights

Effect of external high-dose rate radiation on mouse biofluid metabolomic signatures

Not available

ORGANISM(S): Mus musculus

ST002175 | MetabolomicsWorkbench

Effect of external high-dose rate radiation on mouse biofluid metabolomic signatures (neg)

Not available

ORGANISM(S): Mus musculus

ST002176 | MetabolomicsWorkbench

NMR based quantitative metabolomics studies of heart and lung from mouse exposed to whole body high dose gamma and proton radiation (part II)

Not available

ORGANISM(S): Mus musculus

ST000480 | MetabolomicsWorkbench

Study of molecular mechanisms in lung injury due to ultra-high and conventional dose rate pulsed radiation based on 4D-DIA proteomics

Objective: To investigate the mechanism of ultra-high dose rate pulsed radiation in radiation-induced lung injury (RILI), providing an experimental and theoretical basis for the application of FLASH-RT in radiotherapy. Methods: C57BL/6J mice were randomly divided into three groups: a sham group, a F...

ORGANISM(S): Mus musculus (Mouse)

2025-05-07 | PXD057784 | Pride

Gene expression response to high radiation treatment in MCF7 breast cancer cell line

Intraoperative radiotherapy (IOERT) is a high radiation therapeutic technique which administers a single high dose of ionizing radiation (IR) immediately after surgical tumor removal in order to destroy the residual cancer cells in the site at high risk for recurrence. IR is able to regulate several...

ORGANISM(S): Homo sapiens

2015-06-11 | E-GEOD-63667 | biostudies-arrayexpress

Radiation response in human cardiac endothelial cells investigated by DIA-MS

The endothelium is the barrier separating blood and tissue. Radiation-induced enhanced inflammation leading to permeability of this barrier may increase the risk of cardiovascular disease. The aim of this study was to investigate the onset of endothelial inflammatory pathways after radiation exposur...

ORGANISM(S): Homo sapiens (Human)

2020-11-02 | PXD020735 | Pride

Ionizing radiation Proteomics Ddb2 Inflammation Endothelial cells Cgas-sting-pathway Data-independent acquisition Stat1

Transcription profiling of human skin fibroblasts to low and high doses of UVC radiation to assess cellular transcriptional responses to UVC-induced DNA damage

DNA damage caused by UV radiation initiates cellular recovery mechanisms, which involve activation of DNA damage response pathways, cell cycle arrest and apoptosis. To assess cellular transcriptional responses to UVC-induced DNA damage we compared time course responses of human skin fibroblasts to l...

ORGANISM(S): Homo sapiens

2007-09-21 | E-GEOD-713 | biostudies-arrayexpress

Chronic exposure of humans to high level natural background radiation leads to robust expression of protective stress response proteins

Exposures to low doses of ionizing radiation are relevant since most environmental, diagnostic radiology and occupational exposures lie in this region. However, the molecular mechanisms that drive cellular responses at these doses, and the subsequent health outcomes, remain unclear. A local monazite...

ORGANISM(S): Homo sapiens (Human)

2021-01-28 | PXD022380 | Pride

Chronic low dose radiation Stress response High level natural radiation area (hlnra) Itraq Dose response analysis

Extracellular miRNAs in serum and feces of mice exposed to high-dose radiation

PRJNA1040859 | ENA

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