Cgas-sting-pathway

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Institute of Biochemistry II, Faculty of Medicine, Goethe University Frankfurt; Frankfurt, Germany. (4)
CRCI2NA, Nantes University, INSERM, CNRS, Université d’Angers, Nantes, France. (2)
Dana-Farber Cancer Institute and Harvard Medical School (2)
Max-Planck-Institute for Biology of Ageing Department of Mitochondrial Proteostasis Joseph-Stelzmann-Str. 9b 50931 Cologne (2)
Army medical university (1)
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Cancer Research Center, School of Medicine, Xiamen University (1)
Cancer Science Institute of Singapore, National University of Singapore, Singapore (1)
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Department of Biochemistry and Molecular Biology, Infection and Immunity theme, Biomedicine Discovery Institute, Monash University, Clayton, VIC, Australia (1)
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Division of Cancer Epigenomics, German Cancer Research Center (DKFZ) (1)
Division of Cancer Epigenomics, German Cancer Research Center (DKFZ), INF280, 69120 Heidelberg, Germany (1)
Experimental Systems Immunology, Max Planck Institute of Biochemistry, Martinsried, Germany. (1)
German Cancer Research Center, Division of Cancer Epigenomics (1)
Global Health Institute EPFL Switzerland (1)
Guangdong Cardiovascular Institute, Medical Research Institute, Guangdong Provincial Geriatrics Institute, Guangdong Provincial People’s Hospital (Guangdong Academy of Medical Sciences), Southern Medical University (1)
Head of the Crick Mass Spectrometry Proteomics Platform (1)
Institute for Radiation Biology, Helmholtz Center Munich, German Research Center for Environmental Health GmbH, Neuherberg, Germany (1)
Institute of Biological Chemistry (1)
Medical School of Nanjing University (1)
Novartis Institutes for BioMedical Research, Basel, Switzerland (1)
Peking University (1)
Peking University Health Science Center (1)
Team SOAP, CRCI2NA, Nantes University, INSERM, CNRS, Université d'Angers, Nantes, France. Equipe Labellisée Ligue Contre le Cancer, Nantes (1)
The Institute of Cancer research (1)
University of California San Francisco (1)
University of Oxford (1)
Université de Montréal (1)

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The Kinase CK1a coordinates Initiation and Termination of the cGAS-STING Pathway

The cGAS-STING pathway is an evolutionarily conserved antimicrobial defense mechanism that senses cytosolic DNA to trigger innate immune responses. cGAS and STING play dual roles in tumorigenesis, promoting antitumor immunity and cell death while fueling tumor growth and metastasis. However, the mec...

ORGANISM(S): Homo sapiens (Human)

2026-09-10 | PXD063855 | Pride

cGAS-STING Pathway Mediation in Osteoclast Function and Bone Fracture Healing

PRJNA1328658 | ENA

Urolithin A is a potent modulator of cGAS-Sting signaling pathway

PRJNA930416 | ENA

Intracellular bacteria engage a novel STING-TBK1-MVB12b pathway to enable paracrine cGAS-STING signaling

The innate immune system is crucial for eventual control of infections, but may also contribute to pathology. Listeria monocytogenes is an intracellular gram-positive bacteria and a major cause of food-borne disease. However, important knowledge on the interactions between L. monocytogenes and the i...

ORGANISM(S): Mus musculus (Mouse)

2018-12-20 | PXD012135 | Pride

Phosphoproteomics Sting Cgas Intracellular bacteria Nucleic acid sensing

cGAS-STING pathway in cancer biotherapy.

Not available

S-EPMC7472700 | biostudies-literature

NAD+ supplementation normalizes neuroinflammation and senescence via the cGAS-STING pathway

PRJNA560913 | ENA

Role of cGAS-STING pathway in aging and sexual dimorphism in diabetic kindey disease

PRJNA1016422 | ENA

Attenuating AAV-Triggered Innate Immunity in the Adult Mouse Nervous System via cGAS-STING Pathway Inhibition

PRJNA1369968 | ENA