Ifn-signalling - OmicsDI

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UNIPROT ID/AC

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CHEBI ID

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Metabolite Name

Creatine (9)
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IsPrivate

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First Public Date

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Modelling Approach

Ordinary differential equation model (3)

Study type

Transcription profiling by array (532)
RNA-seq of coding RNA (66)
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ChIP-seq (15)
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Release Date

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2002 (2)
2003 (1)

Lab affiliation

TDI Mass Spectrometry Laboratory, Target Discovery Institute, Nuffield Department of Medicine, University of Oxford, Oxford, Roosevelt Drive, Oxford OX3 7FZ, UK (2)
Biosciences Institute, Faculty of Medical Sciences, Newcastle University, Newcastle upon Tyne, United Kingdom (1)
Biosciences instritute, Newcastle university (1)
Cambridge Institute for Medical Research, University of Cambridge (1)
Center Biomol. Struct. & Organization Head, Immunity and Immune Evasion Lab Bachelor of Health and Biomedical Sciences Honours (BH058) Program Manager School of Health and Biomedical Sciences | RMIT University PO Box 71 | Bundoora 3083 | Australia (1)
Centre for Proteome Research, Dept. Biochemistry & Systems Biology, University of Liverpool, Liverpool, UK (1)
Department of Biochemistry and Chemistry, Centre for Cardiovascular Biology and Disease Research, La Trobe Institute for Molecular Science, School of Agriculture, Biomedicine and Environment, La Trobe University, Bundoora, Victoria 3086, Australia (1)
Department of Clinical Medicine, University of Tromsø 9037 Tromsø, Norway (1)
Global Health Institute EPFL Switzerland (1)
Institute for Microbiology and Infection, School of Biosciences, University of Birmingham, Birmingham B15 2TT UK (1)
Institute of Immunology and Infection Research, Ashworth Laboratories, School of Biological Sciences, University of Edinburgh, EH9 3FL, Edinburgh, United Kingdom (1)
Institute of Immunology and Infection Research, School of Biological Sciences, University of Edinburgh, UK (1)
International Institute of Molecular and Cell Biology, Laboratory of RNA-Protein Interactions - Dioscuri Centre, Warsaw, Poland (1)
MRC Institute of Genetics and Molecular Medicine, Cancer Research UK Edinburgh Centre, The University of Edinburgh ( lab head ) Masaryk Memorial Cancer Institute (1)
MRC Protein Phosphorylation and Ubiquitylation Unit, Sir James Black Centre, Dow Street, School of Life Sciences, University of Dundee, Dundee DD1 5EH, Scotland, UK (1)
Metabolomics and Proteomics Core, Helmholtz zentrum Muenchen (1)
Proteomic Hematology, Department of Experimental Medical Science, Lund Stem Cell Center, Lund University (1)
Ruhr-University Bochum Medizinisches Proteom-Center Clinical Proteomics - Translational Proteomics and University Hospital Knappschaftskrankenhaus Bochum Department of Anesthesia, Intensive Care Medicine, and Pain Therapy (1)
Team Lead in Proteomics Head of the Proteomics Research Translational Technology Platform, UCL Cancer Institute, Paul O’Gorman Building 72 Huntley Street, London, WC1E 6DD, UK (1)
Technical University of Munich (1)
The Institute of Cancer Research, London (1)
The Walter and Eliza Hall Institute of Medical Research, 1G Royal Parade, Melbourne, Victoria 3052, Australia (1)
U o Cambridge (1)
University of Liverpool, Centre for Proteome Research, Biosciences Building, Crown Street, Liverpool, UK (1)
University of Zurich, Institute for Medical Virology, Switzerland (1)

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Chemoresistant Cancer Cell Lines are Characterized by Migratory, Amino Acid Metabolism, Protein Catabolism and IFN1 Signalling Perturbations

Not available

ORGANISM(S): Homo sapiens

ST002104 | MetabolomicsWorkbench

Micropeptide UEIS attenuates cGAS-STING-type I IFN signalling to repress anti-tumour immunity

PRJNA1277268 | ENA

LPS induced CXCL10 chemokine production via IFN- α receptor and toll-like receptor 4 signalling

PRJNA904908 | ENA

Multiomics analysis of neutrophils in SLE: insights from adult and pediatric disease

Neutrophils contribute to systemic lupus erythematosus (SLE) pathogenesis through ROS and NET production, and increased apoptotic debris which causes autoantibody production and immune complex formation. These processes drive inflammation and tissue damage. The aim of this study was to perform in...

2026-04-22 | MTBLS6259 | MetaboLights

IFN-λ4 potently blocks IFN-α signalling by ISG15 and USP18 in hepatitis C virus infection.

Not available

S-EPMC5476576 | biostudies-literature

A co-opted ISG15-USP18 binding mechanism normally reserved for deISGylation controls type I IFN signalling

Type I interferon (IFN) signalling induces the expression of several hundred IFN-stimulated genes (ISGs) that provide an unfavourable environment for viral replication. To prevent an overexuberant response and autoinflammatory disease, IFN signalling requires tight control. One critical regulator is...

ORGANISM(S): Homo sapiens (Human)

2025-03-07 | PXD051575 | Pride