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UnknownGenomic analyses in Cornelia de Lange Syndrome and related diagnoses: Novel candidate genes, genotype-phenotype correlations and common mechanisms.
Not available
S-EPMC10524367 | biostudies-literature
UnknownFavorable lifestyle before diagnosis associated with lower risk of screen-detected advanced colorectal neoplasia.
Not available
S-EPMC4945986 | biostudies-literature
UnknownGenetic heterogeneity in Cornelia de Lange syndrome (CdLS) and CdLS-like phenotypes with observed and predicted levels of mosaicism.
Not available
S-EPMC4173748 | biostudies-literature
TranscriptomicsCircadian Clock Activity in Mouse and Human CD4+ T Cells
Though it is well established that immunological functions of CD4+ T cells are time of day-dependent, the underlying molecular mechanisms remain largely obscure. To address the question whether T cells themselves harbor a functional clock driving circadian rhythms of immune function, we analyzed clo...
ORGANISM(S): Homo sapiens 
2012-04-30 | E-GEOD-29583 | biostudies-arrayexpress
UnknownSynthesis and in vivo evaluation of PEG-BP-BaYbF5 nanoparticles for computed tomography imaging and their toxicity.
Not available
S-EPMC8693452 | biostudies-literature
TranscriptomicsExpression of the placental transcriptome in fetal growth restriction in the Baboon is Dependent on Fetal Sex
Fetal growth restriction (FGR) develops when fetal nutrient availability is compromised and increases the risk for perinatal complications and predisposes for offspring obesity, diabetes and cardiovascular disease later in life. Emerging evidence implicates changes in placental function in altered f...
ORGANISM(S): Papio hamadryas 
2013-12-06 | E-GEOD-40878 | biostudies-arrayexpress
ModelsKrohn2011 - Cerebral amyloid-β proteostasis regulated by membrane transport protein ABCC1
Krohn2011 - Cerebral amyloid-β proteostasis regulated by membrane transport protein ABCC1 This model is described in the article: Cerebral amyloid-β proteostasis is regulated by the membrane transport protein ABCC1 in mice. Krohn M, Lange C, Hofrichter J, Scheffler K, Stenze...
2024-09-02 | BIOMD0000000618 | BioModels
UnknownFully automated dried blood spot sample preparation enables the detection of lower molecular mass peptide and non-peptide doping agents by means of LC-HRMS.
Not available
S-EPMC7220872 | biostudies-literature
ProteomicsHost-to-Pathogen Transfer of Neutrophil Components via Extracellular Vesicles Shields Candida albicans from Immune Attack in Human Blood
Neutrophils effectively eliminate Candida albicans from human blood, but a subset of C. albicans escapes their activity and remains extracellular and viable. Here we show that this is independent from known immune-escape traits of this fungal pathogen. Instead, neutrophil-derived extracellular vesic...
ORGANISM(S): Homo sapiens (Human) 
2026-09-03 | PXD070460 | Pride
Candida albicans Immune evasion Neutrophil-derived extracellular vesicles (evs) Bloodstream infection Complement-dependent opsonization 
UnknownT1 mapping performance and measurement repeatability: results from the multi-national T1 mapping standardization phantom program (T1MES).
Not available
S-EPMC7204222 | biostudies-literature
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