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UnknownPre-ribosomal RNA reorganizes DNA damage repair factors in nucleus during meiotic prophase and DNA damage response.
Not available
S-EPMC8888703 | biostudies-literature
UnknownHuman amniotic mesenchymal stem cells-conditioned medium protects mice from high-fat diet-induced obesity.
Not available
S-EPMC8235646 | biostudies-literature
TranscriptomicsReprogramming fibroblasts into bi-potential hepatic stem cells by defined factors
Recent studies have demonstrated direct reprogramming of fibroblasts into a range of somatic cell types, but to date stem/progenitor cells have only been reprogrammed for the blood and neuronal lineages. We previously reported generation of induced hepatocyte-like (iHep) cells by transduction of Gat...
ORGANISM(S): Mus musculus 
2013-07-19 | E-GEOD-48486 | biostudies-arrayexpress
GenomicsGenome-Wide Association Studies in Upper Gastrointestinal Cancers (Asian)

This project aims to conduct genome-wide association studies (GWAS) in two anatomically different upper gastrointestinal (UGI) cancer sites in two populations with distinctly different disease rates and genetic profiles. One population has very high rates for both esophageal squamous cel...

phs000361.v1.p1 | EGA
UnknownSNX16 aggravates AngII-induced cardiac hypertrophy in mice via EGFR transactivation.
Not available
S-EPMC12770542 | biostudies-literature
ProteomicsProteome landscape of mouse oocyte mitochondria
Mitochondria are essential organelles that provide energy for mammalian cells, with multiple functions such as signal transduction, cellular differentiation, and regulation of apoptosis. Compared with the mitochondria in somatic cells, oocyte mitochondria have an additional layer of importance since...
ORGANISM(S): Mus Musculus 
2021-03-16 | PXD024777 |
UnknownSPINK1, PRSS1, CTRC, and CFTR Genotypes Influence Disease Onset and Clinical Outcomes in Chronic Pancreatitis.
Not available
S-EPMC6232107 | biostudies-literature
ProteomicsEpididymal epithelial degeneration and lipid metabolism impairment account for male infertility in occludin knockout mice
Occludin (OCLN) is a tight junction protein and Ocln deletion mutation causes male infertility in mice. However, the role of OCLN in male reproductive system remains unknown. In this study, we found defective proximal epididymis in Ocln-null mice with impaired sperm mobility and fertilization, but n...
ORGANISM(S): Mus Musculus 
2022-10-25 | PXD037690 |
ProteomicsProteome from whole epididymis and caput epididymidis of WT and Ocln knockout mice
Epithelial tight junction is the frontline barrier for physiological homeostasis and protection of a host and Occludin (Ocln) is a necessary component of the tight junction complex. Male Ocln knockout (Ocln-KO) mice are overall normal but infertile. The cause of this phenotype remains elusive. We fo...
ORGANISM(S): Mus Musculus 
2021-02-05 | PXD024008 |
UnknownRoles of blood monocytes carrying TREM2<sup>R47H</sup> mutation in pathogenesis of Alzheimer's disease and its therapeutic potential in APP/PS1 mice.
Not available
S-EPMC11848385 | biostudies-literature
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