Marusiak Anna A

Show results for

Proteomics (2)
Transcriptomics (1)

Organisms

Alphavirus (2)
Anemia (2)
Archaea (2)
Bacteria (2)
Betaherpesvirinae (2)
Borrelia (2)
Clostridium perfringens (2)
Cytomegalovirus (2)
Dengue virus (2)
Eukaryota (2)
Hepatitis B virus (2)
Human alphaherpesvirus 1 (2)
Human betaherpesvirus 5 (2)
Human immunodeficiency virus (2)
Influenza A virus (2)
Influenza C virus (2)
Japanese encephalitis virus group (2)
Kamptonema sp. (2)
Listeria monocytogenes (2)
Mammarenavirus (2)
Miscanthus x giganteus (2)
Mycobacterium tuberculosis (2)
Orthoflavivirus (2)
Pseudomonas aeruginosa (2)
Rattus (2)
Sarbecovirus (2)
Severe acute respiratory syndrome coronavirus 2 (2)
Staphylococcus aureus (2)
Streptococcus pneumoniae (2)
Homo sapiens (1)

Repository

pride (2)
biostudies-arrayexpress (1)

Tissue

Cell culture (1)
Head and neck squamous cell carcinoma cell line (1)

Disease

Breast cancer (1)

Technology Type

Bottom-up proteomics (2)
Data-dependent acquisition (2)
Mass Spectrometry (2)

Publication Date

2014 (1)
2025 (1)
2024 (1)

Study type

Transcription profiling by array (1)

Release Date

2014 (1)

Lab affiliation

Laboratory of Molecular OncoSignalling, IMol Polish Academy of Sciences, Warsaw, Poland (1)
The International Institute of Molecular Mechanisms and Machines, Polish Academy of Sciences (1)

Previous page
1 / 1
You're on page 1
Next page

Sort by:

Relevance

Page size

Identification of protein substrates of a novel proteolysis targeting chimera, PROTAC124 in tongue squamous cell carcinoma cell line CAL-33 overexpressing Leucine Zipper-bearing Kinase (LZK).

Head and neck squamous cell carcinomas (HNSCC) are aggressive epithelial tumors with a low and unchanged 50% 5-year survival rate. MAP3K13 gene encoding Leucine Zipper-bearing Kinase (LZK) is often amplified in HNSCC patients and can be an oncogenic driver for tumorigenesis. In order to reduce catal...

ORGANISM(S): Homo sapiens (Human)

2025-06-16 | PXD057778 | Pride

Frequent Loss-of-Function Mutations in MLK4 Suppresses Signaling in the JNK-cJUN-p21/p15 Pathway to Promote Growth of Colon Cancer Cells

The goal of the study was to evaluate the influence of mutations in MLK4 on the protein function and the process of tumorigenesis in colorectal cancers. Biochemical data imply that a majority of MLK4 mutations in colon cancer are loss-of-function, including, E314K and Y330H mutations. Two colorectal...

ORGANISM(S): Homo sapiens

2014-07-09 | E-GEOD-59196 | biostudies-arrayexpress

The effect of an MLK3-targeting PROTAC on protein levels in model breast cancer cell lines.

The objective of the study was to explore how a new compound (CEP1347-VHL-02 PROTAC) which facilitates the breakdown of mitogen-activated protein kinase kinase kinase 11 (MLK3), affects the protein quantities in the total cell extracts from specific breast cancer cell models. The cell lines used in ...