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ProteomicsMAZ induces MYB expression during the exit from quiescence via the E2F site in the MYB promoter
Most E2F-binding sites repress transcription through the recruitment of Retinoblasoma (RB) family members until the end of the G1 cell-cycle phase. Although the MYB promoter contains an E2F-binding site, its transcription is activated shortly after the exit from quiescence, before RB family members ...
ORGANISM(S): Homo sapiens (Human) 
2017-10-04 | PXD006857 | Pride
Quiescence Retinoblasoma Myb Maz 
ProteomicsZinc-dependent Zip7-MAZ-MYBL2 axis promotes prostate cancer metastasis
Screening reveals that a zinc transporter Zip7 was upregulated in metastatic PCa and related to poor progression. Zip7 silencing inhibits PCa cell migration and invasion in vitro and bone-metastasis in vivo. Mechanistically, we identify that Zip7 mainly interacts with MAZ in cytoplasm to facilita...
ORGANISM(S): Homo Sapiens 
2026-01-14 | PXD073047 |
GenomicsGlobal transcriptions upon MAZ knockdown HUDEP-2 cells
Global transcriptions upon MAZ knockdown HUDEP-2 cells
PRJNA1184152 | ENA
GenomicsCRISPR and biochemical screens identify MAZ as a co-factor in CTCF-mediated insulation at Hox clusters [MAZ KO]
CRISPR and biochemical screens identify MAZ as a co-factor in CTCF-mediated insulation at Hox clusters [MAZ KO]
PRJNA660249 | ENA
GenomicsGenome-wide map of MAZ transcription factor in primary human erythroid cells
Genome-wide map of MAZ transcription factor in primary human erythroid cells
PRJNA579097 | ENA
GenomicsCRISPR and biochemical screens identify MAZ as a co-factor in CTCF-mediated insulation at Hox clusters [MAZ motif deletion RNA-seq]
CRISPR and biochemical screens identify MAZ as a co-factor in CTCF-mediated insulation at Hox clusters [MAZ motif deletion RNA-seq]
PRJNA660250 | ENA
TranscriptomicsGlobal transcriptions upon MAZ knockdown HUDEP-2 cells
Hemoglobinopathies, such as sickle cell disease and β-thalassemia, are common genetic disorders remaining significant global health challenges due to their associated morbidity and mortality. Increasing fetal hemoglobin (HbF) levels has emerged as a promising therapeutic strategy for these disorders...
ORGANISM(S): Homo sapiens 
2025-11-01 | GSE281501 | GEO
UnknownMAZ regulates fetal hemoglobin repression by activating MYB transcription.
Not available
S-EPMC12783104 | biostudies-literature
UnknownPUF60-Regulated Isoform Switching of MAZ Modulates Gastric Cancer Cell Migration.
Not available
S-EPMC12102611 | biostudies-literature
GenomicsCRISPR and biochemical screens Identify MAZ as a co-factor in CTCF-mediated insulation at Hox Clusters
CRISPR and biochemical screens Identify MAZ as a co-factor in CTCF-mediated insulation at Hox Clusters
PRJNA789256 | ENA
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