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TranscriptomicsTranscription profiling of human MCF-7 cells treated with proteasome inhibitor MG132 and either dexamethasone or 17b-estradiol
The objective of the study was to investigate the effect of proteasome inhibition on glucocorticoid and estrogen receptor regulated gene expression. Experiment Overall Design: MCF-7 cells were treated with proteasome inhibitor (MG132), dexamethasone, 17b-estradiol or MG132 plus dexamethasone or MG13...
ORGANISM(S): Homo sapiens 
2008-06-20 | E-GEOD-8383 | biostudies-arrayexpress
TranscriptomicsCCRF-CEM Prednisolone and MG132 treatments at 4h
Background: The combination of Proteasome inhibitor with Glucocorticoid is one of the most promising antileukemic treatments. Both agents act through pluripotent signal mediators. The two types of agents inhibit key signals that are considered crucial to their effects on malignant cells. Methodology...
ORGANISM(S): Homo sapiens 
2011-05-11 | E-GEOD-29136 | biostudies-arrayexpress
TranscriptomicsGene expression profiling of Glioma Stem Cells (GSCs) and their differentiated cell counterparts after MG132 treatment
Analysis of genes in GSCs and differentiated cells that are induced by MG132 treatment. Total RNAs were isolated from GSCs and differentiated cells after MG132 or DMSO treatment for 6 hrs in serum-free media
ORGANISM(S): Homo sapiens 
2014-10-15 | E-GEOD-62356 | biostudies-arrayexpress
TranscriptomicsSystemic Comparison of Heat Shock Response Induced by Heat Shock and a Proteasome Inhibitor in Mouse Fibrosarcoma Cells and Their Thermotolerant Counterparts
Heat shock response (HSR) is a cellular defense mechanism against various stresses. Both heat shock and proteasome inhibitor MG132 cause the induction of heat shock proteins, a distinct feature of HSR. To better understand the molecular basis of HSR, we subjected the mouse fibrosarcoma cell line, RI...
ORGANISM(S): Mus musculus 
2011-11-09 | E-GEOD-24197 | biostudies-arrayexpress
ProteomicsSILAC proteomics on MG132-induced nuclear aggregates
H1299 cells were labelled either with light (Lys0/Arg0) or heavy (Lys8/Arg10) amino acids and were either unstressed (light), stressed (heavy) with MG132 (15hrs) or stressed with MG132 and allowed to recover for 8hrs (light). Equal number of unstressed and MG132 stressed cells or MG132 stressed cell...
ORGANISM(S): Homo sapiens (Human) 
2025-05-07 | PXD054637 | Pride
Nucleolus Nuclear inclusions Silac Proteasome Mg132 
Proteomicsquantitative proteomic analysis of proteasome in response to MG132
The ubiquitin-proteasome system and autophagy are two major intracellular proteolytic pathways and both remove misfolded and proteotoxic proteins from eukaryotic cells. This study describes the detailed informations about the change in composition and interactome of proteasomes after prolonged MG132...
ORGANISM(S): Homo sapiens (Human) 
2021-09-09 | PXD019193 | Pride
Proteasome Interactome Mg132 
GenomicsThe proteasome inhibitor MG132 adjusts the transcriptome in Brassica napus roots
The proteasome inhibitor MG132 adjusts the transcriptome in Brassica napus roots
PRJNA525819 | ENA
GenomicsExpression data from NRF1 knockdown and/or MG132 treated HCT116 cells
Expression data from NRF1 knockdown and/or MG132 treated HCT116 cells
PRJNA944192 | ENA
TranscriptomicsEffect of MG132 on expression profile of Schistosoma mansoni adult worms
Transcriptional profiling of MG132 treated S. mansoni adult worms (50uM of MG132 in DMSO during 24h) vs. Non-treated S.mansoni adult worms (DMSO during 24h) Two conditions (treated vs. Non-treated) with four biological samples of treated worms (MG132 1, 2, 3 and 4) and four biological samples of non...
ORGANISM(S): Schistosoma mansoni 
2014-12-16 | GSE57722 | GEO
GenomicsChromatin state of MCF-7 breast cancer cells treated with proteasome inhibitor MG132 [histone_mod_chip_seq]
Chromatin state of MCF-7 breast cancer cells treated with proteasome inhibitor MG132 [histone_mod_chip_seq]
PRJNA595567 | ENA
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