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MetabolomicsMetabolomics profiling reveals changes in peptidoglycan and redox metabolism between ancient and modern Mycobacterium tuberculosis

Mycobacterium tuberculosis (Mtb), the cause of tuberculosis, kills over one million people worldwide each year. Mtb is classified into ten distinct lineages, each harboring unique genetic changes that influence its survival, virulence, and transmissibility. Although metabolism plays a cr...

2026-06-26 | MTBLS14029 | MetaboLights
TranscriptomicsExpression data from peptidoglycan stimulated CD11b+ cells from NOD and NOD.Idd3
CD11b+ CD11c- cells were isolated by cell sorting from the spleens of NOD and NOD.Idd3 mice. Cells were then either unstimulated or stimulated with peptidoglycan (5ug/ml) for 4 hours at 37 degrees prior to RNA extraction. Peptidoglycan was chosen as a stimulus due to its know ability to stimulate C...
ORGANISM(S): Mus musculus 
2010-09-28 | E-GEOD-24350 | biostudies-arrayexpress
TranscriptomicsTranscriptomic profiles of two sponge species upon exposure to microbial-associated molecular patterns (lipopolysaccharide and peptidoglycan) compared against control (sterile artificial seawater)
The interaction of animals with microbes relies on the specific recognition of microbial-derived molecules by receptors of the immune system. Sponges (phylum Porifera), as sister group of the Eumetazoa, provide insights into conserved mechanisms for animal-microbe crosstalk, but empirical data is li...
ORGANISM(S): Dysidea avara 
2018-11-26 | E-MTAB-6757 | biostudies-arrayexpress
ProteomicsArchaea produce peptidoglycan hydrolases that kill bacteria
The social life of archaea is poorly understood. In particular, even though competition and conflict are common themes in microbial communities, there is scant evidence documenting antagonistic interactions between archaea and their abundant prokaryotic brethren: bacteria. Do archaea specifically ta...
ORGANISM(S): Haloferax volcanii SB1 
2026-02-07 | PXD064732 | Pride
Archaea Peptidoglycan hydrolase 
GenomicsResponse of Vibrio cholerae to exogenous peptidoglycan
Response of Vibrio cholerae to exogenous peptidoglycan
PRJNA894977 | ENA
ProteomicsS. aureus peptidoglycan hydrolysis by mutanolsyin
(Manuscript abstract) "Peptidoglycan, a gigadalton polymer, functions as the scaffold for bacterial cell walls and provides cell integrity. Peptidoglycan is remodelled by a large and diverse group of peptidoglycan hydrolases, which control bacterial cell growth and division. Over the years, many stu...
ORGANISM(S): Staphylococcus aureus 
2023-11-06 | PXD045856 | Pride
Peptidoglycan Staphylococcus aureus Mutanolysin 
TranscriptomicsEffect of Drosophila immune proteins on gene expression in E. coli
Interaction between the host and invading pathogen determines the fate of both organisms during the infectious state. The host is equipped with a battery of immune reactions, while the pathogen displays a variety of mechanisms to compromise host immunity. Although bacteria alter their pattern of g...
ORGANISM(S): Escherichia coli 
2015-01-13 | E-GEOD-61604 | biostudies-arrayexpress
UnknownPlant peptidoglycan precursor biosynthesis: Conservation between moss chloroplasts and Gram-negative bacteria.
Not available
S-EPMC9434261 | biostudies-literature
ProteomicsGenetic and pharmacological inactivation of peptidoglycan remodeling increases antibiotic susceptibility of vancomycin-resistant Enterococcus faecium
Vancomycin-resistant Enterococcus faecium (VREfm) is a leading cause of healthcare-associated infections globally and demands new approaches for treatment. Here we show that genetic and pharmacological inactivation of a highly conserved NlpC/P60 peptidoglycan hydrolase, secreted antigen A (SagA), en...
ORGANISM(S): Enterococcus faecium ERV165 
2026-04-13 | PXD075040 | Pride
Antibiotic adjuvant Covalent inhibitor Chemoproteomics Vancomycin-resistant enterococcus faecium Peptidoglycan hydrolase 
GenomicsMurA escape mutations uncouple peptidoglycan biosynthesis from PrkA signaling
MurA escape mutations uncouple peptidoglycan biosynthesis from PrkA signaling
PRJEB47255 | ENA
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