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TranscriptomicsDeleterious assembly of the lamin A/C mutant p.S143P causes ER stress in familial dilated cardiomyopathy
Mutation of the LMNA gene, encoding nuclear lamin A and lamin C (hereafter lamin A/C), is a common cause of familial dilated cardiomyopathy (DCM). Among Finnish DCM patients, the founder mutation c.427T>C (p.S143P) is the most frequently reported genetic variant. Here, we show that p.S143P lamin A/C...
ORGANISM(S): Homo sapiens 
2016-06-10 | E-GEOD-82290 | biostudies-arrayexpress
Transcriptomics5'RNA-seq analysis of hypertrophic cardiomyopathy (HCM) in mice that carry a human missence mutation in the myosin heavy chain
We developed a 5'RNA-seq methodology to concurrently assess gene expression and start-site usage changes. We applied this methodology to study hypertrophic cardiomyopathy in mice harboring a human deleterious mutation. 5'RNA-seq analysis of transcriptomes from mouse hearts with or without hypertroph...
ORGANISM(S): Mus musculus 
2013-11-05 | E-GEOD-52038 | biostudies-arrayexpress
ProteomicsProteomic of Stress-Induced Cardiomyopathy
Label free quantitative proteomics method was used to analyze protein abundance under defined conditions.
ORGANISM(S): Mus Musculus 
2024-09-26 | PXD056309 |
ProteomicsConstitutive Protein Kinase G Activation Exacerbates Stress-induced Cardiomyopathy
: Heart failure is associated with high morbidity and mortality, and new therapeutic targets are needed. Preclinical data suggest that pharmacological activation of protein kinase G (PKG) can reduce maladaptive ventricular remodeling and cardiac dysfunction in the stressed heart. However, clinical...
ORGANISM(S): Mus musculus (Mouse) 
2023-07-20 | PXD022600 | Pride
Lc-msms Protein kinase g 
UnknownStress-Induced Cardiomyopathy Presenting as Shock.
Not available
S-EPMC4828422 | biostudies-literature
UnknownRare Variants in Cardiomyopathy Genes Associated With Stress-Induced Cardiomyopathy.
Not available
S-EPMC4871739 | biostudies-literature
ProteomicsEarly Cardiac Proteomic Profiling in a Long-Latency Doxorubicin Cardiomyopathy Model Exhibits Sexual Dimorphism for Cardioprotection
In this study, we employed large-scale data-independent acquisition (DIA) proteomics in a long-latency rat model of doxorubicin-induced cardiomyopathy to characterize early cardiac proteomic remodeling prior to overt functional decline. Male and female Sprague-Dawley rats received a cumulative low d...
ORGANISM(S): Rattus norvegicus (Rat) 
2026-09-14 | PXD076815 | Pride
Doxorubicin Dia Proteomics Female Long latency Male Data-independent acquisition Cardiomyopathy 
UnknownMagnetocardiography in Diagnosis of Stress-Induced Cardiomyopathy.
Not available
S-EPMC12441464 | biostudies-literature
UnknownStress-Induced Cardiomyopathy Secondary to COVID-19.
Not available
S-EPMC7479482 | biostudies-literature
UnknownStress-Induced Cardiomyopathy Following Kidney Transplant.
Not available
S-EPMC12441348 | biostudies-literature
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