Sort   by:  
 Page size 
MetabolomicsDisruption of the Brain-Spleen Axis Impairs Monocyte-Microglia Communication and Accelerates Disease Progression in a Mouse Model of Amyloidosis
Alzheimer’s disease (AD) is characterized by a prolonged asymptomatic phase before cognitive decline emerges, yet the mechanisms driving symptom onset remain unclear. Here, we hypothesized that the transition from asymptomatic to symptomatic disease is linked to dysfunction of brain–immune communica...
2026-06-01 | MTBLS14615 | MetaboLights
UnknownSynthesis and surface spectroscopy of α-pinene isotopologues and their corresponding secondary organic material.
Not available
S-EPMC6844218 | biostudies-literature
UnknownOptoacoustic Imaging in Inflammation.
Not available
S-EPMC8145174 | biostudies-literature
UnknownGiant photoluminescence enhancement in MoSe2 monolayers treated with oleic acid ligands.
Not available
S-EPMC8276785 | biostudies-literature
MetabolomicsMetabolostasis failure thresholds are linked with network topology, metabolite solubility, and translational control

Cells maintain metabolite homeostasis (metabolostasis) by buffering fluctuations in metabolite levels, yet the limits of this buffering and the mechanisms underlying metabolic toxicity remain poorly understood. To study this, we systematically overfed metabolites in Saccharomyces cerevisiae and q...

2026-09-29 | MTBLS12080 | MetaboLights
MetabolomicsRapid UHPLC-MS metabolite profiling and phenotypic assays reveal genotypic impacts of nitrogen supplementation in oats
INTRODUCTION: Oats (Avena sativa L.) are a whole grain cereal recognised for their health benefits and which are cultivated largely in temperate regions providing both a source of food for humans and animals, as well as being used in cosmetics and as a potential treatment for a number of...
2020-06-11 | MTBLS804 | MetaboLights
UnknownEnhancing Photoluminescence and Mobilities in WS2 Monolayers with Oleic Acid Ligands.
Not available
S-EPMC6746058 | biostudies-literature
ProteomicsDysfunction of a SET3C-like complex underlies a family of related neurological disorders
TBLR1, a subunit of the NCoR corepressor complex, is mutated in a range of neurodevelopmental disorders that only partially align symptomatically with those caused by mutations in its binding partner, MeCP2. In a search for novel TBLR1-binding partners, we identified ANKRD11 and SETD5 – mutations in...
ORGANISM(S): Homo sapiens (Human) 
2026-05-01 | PXD077916 | Pride
Dia. Tblr1 Mecp2 Set3c-like complex Ankrd11 Setd5 Neurodevelopmental disorders Dda 
ProteomicsDysfunction of a SET3C-like complex underlies a family of related neurological disorders
TBLR1, a subunit of the NCoR corepressor complex, is mutated in a range of neurodevelopmental disorders that only partially align symptomatically with those caused by mutations in its binding partner, MeCP2. In a search for novel TBLR1-binding partners, we identified ANKRD11 and SETD5 – mutations in...
ORGANISM(S): Mus musculus (Mouse) 
2026-05-01 | PXD063846 | Pride
Human Tblr1 Neurological disorders Ankrd11 Setd5 Set3c 
UnknownRetinal pigment epithelium transplantation: concepts, challenges, and future prospects.
Not available
S-EPMC4541358 | biostudies-literature
Sort   by:  
 Page size