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ProteomicsAnalysis of FAM65A interactions
We identified FAM65A as a novel interactor of RHOA. To reveal other binding partners of FAM65A, we carried out a quantitative IP-MS, using SILAC labelled HeLa cells ectopically expressing GFP-FAM65A or GFP alone as control. Experiments were performed in duplicate with switched labelling and specific...
ORGANISM(S): Homo sapiens (Human) 
2016-11-01 | PXD004933 | Pride
Ip-ms  silac  fam65a  rho 
UnknownFAM65A promotes the progression and growth of lung squamous cell carcinoma in vivo and vitro.
Not available
S-EPMC11295694 | biostudies-literature
UnknownFAM65A as a novel prognostic biomarker in human tumors reveal by a pan-cancer analysis.
Not available
S-EPMC8777545 | biostudies-literature
UnknownFAM65A, as a potential predictor of prognosis, promotes colorectal cancer progression via activating Ras/ERK/RSK signaling.
Not available
S-EPMC12874459 | biostudies-literature
Methylation profilingGenome-Wide DNA Methylation Profiling of the Failing Human Heart with Mechanical Unloading Identifies LINC00881 as an Essential Regulator of Calcium Handling in the Cardiomyocyte [DNA Methylation]
Background: Human heart failure is characterized by global alterations in the myocardial DNA methylation profile, yet little is known about epigenetic regulation of non- coding transcripts and potential reversibility of DNA methylation with left ventricular assist device (LVAD) support. Method: High...
ORGANISM(S): Homo sapiens 
2022-03-04 | GSE197670 | GEO
TranscriptomicsGenome-Wide DNA Methylation Profiling of the Failing Human Heart with Mechanical Unloading Identifies LINC00881 as an Essential Regulator of Calcium Handling in the Cardiomyocyte [RNAseq]
Background: Human heart failure is characterized by global alterations in the myocardial DNA methylation profile, yet little is known about epigenetic regulation of non- coding transcripts and potential reversibility of DNA methylation with left ventricular assist device (LVAD) support. Method: High...
ORGANISM(S): Homo sapiens 
2022-03-04 | GSE197671 | GEO
ProteomicsAnalysis of total-proteome change following MST3/4 depletion
To assess changes in protein expression levels following loss of MST3/4, we co-depleted MST3 and MST4 and quantified the changes in total protein levels by using SILAC.
ORGANISM(S): Homo sapiens (Human) 
2022-02-28 | PXD006168 | Pride
Mst3  mst4  ccm3  gckiii  fam65a 
ProteomicsAnalysis of phospho-proteome change following MST3/4 depletion
Mammalian STE20-like protein kinases 3 and 4 (MST3/4) have recently emerged as crucial regulators of several key cellular processes such as proliferation, migration, and polarity. However, the downstream target pathways which are regulated by the kinase activity of MST3/4 remain poorly characterised...
ORGANISM(S): Homo sapiens (Human) 
2022-02-28 | PXD006167 | Pride
Mst3  mst4  ccm3  gckiii  fam65a  stripak 
GenomicsGenome-Wide DNA Methylation Profiling of the Failing Human Heart with Mechanical Unloading Identifies LINC00881 as an Essential Regulator of Calcium Handling in the Cardiomyocyte [RNAseq]
Genome-Wide DNA Methylation Profiling of the Failing Human Heart with Mechanical Unloading Identifies LINC00881 as an Essential Regulator of Calcium Handling in the Cardiomyocyte [RNAseq]
PRJNA811541 | ENA
GenomicsGenome-Wide DNA Methylation Profiling of the Failing Human Heart with Mechanical Unloading Identifies LINC00881 as an Essential Regulator of Calcium Handling in the Cardiomyocyte [DNA Methylation]
Genome-Wide DNA Methylation Profiling of the Failing Human Heart with Mechanical Unloading Identifies LINC00881 as an Essential Regulator of Calcium Handling in the Cardiomyocyte [DNA Methylation]
PRJNA811540 | ENA
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