Sort   by:  
 Page size 
TranscriptomicsDifferential gene expression profiling of MIHA cells transfected with C-terminal truncated HBx mutants
To compare the differentially expressed transcriptomes between MIHA cells transfected with empty vector control or different C-terminal truncated HBx mutants (14 or 35 amino acid carboxyl-terminal truncation - i.e. d14 and d35) mRNA profiles of MIHA cells stably overexpressing empty vector control o...
ORGANISM(S): Homo sapiens 
2016-08-02 | E-GEOD-71993 | biostudies-arrayexpress
GenomicsFGFR2 C-terminal truncation in cancer
FGFR2 C-terminal truncation in cancer
PRJEB42514 | ENA
Genomics
RNA sequencing for CAP1 mutant with C-terminal truncation
PRJNA1299481 | ENA
TranscriptomicsTranscription profiling of yeast Med2, Med3, Med18, Med20 deletion mutants and with a C-terminal truncation of Med8
We made deletions of Med2, Med3, Med18, Med20 and a C-terminal truncation of Med8. We expression profiled the mutants against a common reference.
ORGANISM(S): Saccharomyces cerevisiae 
2008-03-08 | E-MEXP-1341 | biostudies-arrayexpress
ProteomicsLow-expressing synucleinopathy mouse models based on oligomer formation and C-terminal truncation of α-synuclein
Two mouse models were developed that expressed mutated human alpha synuclein under the control of endogenous murine αSyn (mαSyn) regulatory regions. The expression of the mαSyn was thereby abolished. The genetically ingeneered mice expressed either human αSyn (hαSyn) with a C-terminal deletion endin...
ORGANISM(S): Mus musculus (Mouse) Homo sapiens (Human) 
2021-05-28 | PXD022314 | Pride
Α-synuclein mutants Neuroinflammation Transgenic mouse Truncation Motor impairment Oligomerization Parkinson’s disease Olfaction 
ProteomicsDetection and Quantification of Novel C-terminal TDP-43 Fragments in ALS-TDP
The pathological hallmark of amyotrophic lateral sclerosis (ALS) is the presence of cytoplasmic inclusions, containing C-terminal fragments of the protein TDP-43. Here, we tested the hypothesis that highly sensitive mass spectrometry with parallel reaction monitoring (MS-PRM) can generate a high-res...
ORGANISM(S): Homo sapiens (Human) 
2020-12-17 | PXD022186 | Pride
Prm Tdp-43 Als 
GenomicsPartial truncation of the C-terminal domain of PTCH1 in cancer promotes tumorigenesis by non-canonical activation of GLI transcriptional activity.
Partial truncation of the C-terminal domain of PTCH1 in cancer promotes tumorigenesis by non-canonical activation of GLI transcriptional activity.
PRJNA1245284 | ENA
UnknownExploring the Impact of Physiological C-Terminal Truncation on α-Synuclein Conformations to Unveil Mechanisms Regulating Pathological Aggregation.
Not available
S-EPMC11588551 | biostudies-literature
UnknownC-terminal truncation modulates α-Synuclein's cytotoxicity and aggregation by promoting the interactions with membrane and chaperone.
Not available
S-EPMC9363494 | biostudies-literature
GenomicsThe impact of RNase E C-terminal truncation mutant compared with wildtype RNase E on whole transcriptome expression profile of E. coli soft-agar plate culture
The impact of RNase E C-terminal truncation mutant compared with wildtype RNase E on whole transcriptome expression profile of E. coli soft-agar plate culture
PRJNA1189073 | ENA
Sort   by:  
 Page size