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UnknownMolecular Regulation of Porcine Skeletal Muscle Development: Insights from Research on CDC23 Expression and Function.
Not available
S-EPMC11011816 | biostudies-literature
UnknownRole of Cdc23/Mcm10 in generating the ribonucleotide imprint at the mat1 locus in fission yeast.
Not available
S-EPMC6468313 | biostudies-literature
UnknownData collection with a tailored X-ray beam size at 2.69 A wavelength (4.6 keV): sulfur SAD phasing of Cdc23(Nterm).
Not available
S-EPMC4784671 | biostudies-literature
ProteomicsGlobal Phosphoproteomics of DYRK1A Inhibition in Glioblastoma Cells
Here we performed a detailed investigation to delineate the role of DYRK1A in glioblastoma, using both pharmacological and genetic tools combined with global phosphoproteomics. Our data show that DYRK1A inhibitors affect a much broader proportion of the phosphoproteome than DYRK1A knockdown. By over...
ORGANISM(S): Homo sapiens (Human) 
2021-04-22 | PXD020441 | Pride
Glioblastoma Proteomics U251 Dyrk1a Phosphoproteome Post-translational modification Mass spectrometry Signal transduction Phosphorylation Cancer Phosphoproteomics 
ProteomicsCDK9-55 guides the anaphase-promoting complex/cyclosome (APC/C) in choosing the DNA repair pathway by affecting the UFL1 ubiquitination.
DNA double-strand breaks (DSBs) contribute to genome instability, a key feature of cancer. DSBs are mainly repaired by homologous recombination (HR) and non-homologous end-joining (NHEJ). We investigated the role of an isoform of the multifunctional cyclin-dependent kinase 9, CDK9-55, in DNA repair,...
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2021-07-20 | MSV000087856 | MassIVE
UnknownThe four canonical tpr subunits of human APC/C form related homo-dimeric structures and stack in parallel to form a TPR suprahelix.
Not available
S-EPMC3898896 | biostudies-literature
UnknownAPC/CCDC20 and APC/C play pivotal roles in the process of embryonic development in Artemia sinica.
Not available
S-EPMC5171921 | biostudies-literature
ProteomicsGene expression analysis of yeast strains with a nonsense mutation in the eRF3-coding gene highlights possible mechanisms of adaptation
In yeast Saccharomyces cerevisiae, there are two translation termination factors, eRF1 (SUP45) and eRF3 (SUP35), which are essential for viability. Previous studies have revealed that presence of nonsense mutations in these genes leads to amplification of mutant alleles (sup35-n and sup45-n) which ...
ORGANISM(S): Saccharomyces cerevisiae (Baker's yeast) 
2026-08-17 | PXD052727 | Pride
15 rna-seq Cdc28 Yeast Nonsense mutations Erf3 Cdc23 Translation termination Cell cycle Cdc20 Proteome Fkh1 Release factors 
UnknownPATL2 mutations affect human oocyte maternal mRNA homeostasis and protein interactions in cell cycle regulation.
Not available
S-EPMC11686847 | biostudies-literature
UnknownA novel yeast screen for mitotic arrest mutants identifies DOC1, a new gene involved in cyclin proteolysis.
Not available
S-EPMC25633 | biostudies-literature
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