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ModelsGoldbeter2008_Somite_Segmentation_Clock_Notch_Wnt_FGF
This is a model of the coupled Natch, Wnt and FGF modules as described in: A. Goldbeter and O. Pourquié , Modeling the segmentation clock as a network of coupled oscillations in the Notch, Wnt and FGF signaling pathways. J Theor Biol. 2008 Jun 7;252(3):...
2024-09-02 | BIOMD0000000201 | BioModels
ProteomicsModern Metaproteomics: A Unique Tool to Characterize the Active Microbiome in Health and Diseases, and Pave the Road towards New Biomarkers. Example of Crohn's Disease and Ulcerative Colitis Flare-ups
Thanks to the latest developments in mass spectrometry, software and standards, metaproteomics is emerging as the vital complement of metagenomics, to make headway in understanding the actual functioning of living and active microbial communities. Modern metaproteomics offers new possibilities in th...
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2022-03-22 | MSV000089099 | MassIVE
ProteomicsAnterior Gradient protein 2 is a marker of tumor aggressiveness in breast cancer and favors chemotherapy-induced senescence escape.
Among the different chemotherapies available, genotoxic drugs are widely used. In response to these drugs, particularly doxorubicin, tumor cells can enter into senescence. Chemotherapy-induced senescence (CIS) is a complex response. Long described as a definitive arrest of cell proliferation, we and...
ORGANISM(S): Homo sapiens (Human) 
2022-02-15 | PXD028073 | Pride
Chemotherapy Agr2 Senescence 
ProteomicstRNA Biogenesis and Specific Aminoacyl-tRNA Synthetases Regulate Senescence Stability Under the Control of mTOR
Oncogenes or chemotherapy treatments trigger the induction of suppressive pathways such as apoptosis or senescence. Senescence was initially defined as a definitive arrest of cell proliferation but recent results have shown that this mechanism is also associated with cancer progression and chemother...
ORGANISM(S): Homo sapiens (Human) 
2021-12-09 | PXD029535 | Pride
Chemotherapy Trna Er stress Senescence P21 Mtor 
ProteomicsSARS-CoV-2 Mpro subtiligase-mediated N-terminomics in Jurkat
Subtiligase-based N-terminomics study with 0.5 uM SARS-CoV-2 Mpro in Jurkat cell lysates. Independent duplicates were injected with (L20200924-06, L20200924-07) and without FAIMS (L20200927-01-001, L20200927-02-002).
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2021-12-17 | MSV000088584 | MassIVE
ProteomicsSARS-CoV-2 Mpro subtiligase-mediated N-terminomics in A549
Subtiligase-based N-terminomics study with 0.5 uM of recombinant SARS-CoV-2 Mpro in A549 cell lysates, in duplicate experiments.
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2021-12-17 | MSV000088583 | MassIVE
ProteomicsReverse N-terminomics of SARS-CoV-2 PLpro in A549
Subtiligase based N-terminomics (Abu N-terminal tagging) of two A549 cell lysate replicates incubated with SARS-CoV-2 PLpro.
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2022-08-12 | MSV000090125 | MassIVE
ProteomicsReverse N-terminomics of SARS-CoV-2 PLpro in Jurkat
Subtiligase based reverse N-terminomics (Abu N-terminal tagging) of two Jurkat cell lysate replicates incubated with SARS-CoV-2 PLpro.
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2022-08-12 | MSV000090124 | MassIVE
ModelsGoldbeter2007_Somitogenesis_Switch
This is the simple model without diffusion described in th epublication Sharp developmental thresholds defined through bistability by antagonistic gradients of retinoic acid and FGF signaling. Goldbeter A, Gonze D, Pourquié O. Dev Dyn. 2007 Jun;236(6):1495-508. PMID: 17497689,...
2024-09-02 | BIOMD0000000275 | BioModels
UnknownLow circulating adropin concentrations predict increased risk of cognitive decline in community-dwelling older adults.
Not available
S-EPMC10828274 | biostudies-literature
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